Aortic Stenosis

Aortic Stenosis
1. Etiology
a. Rheumatic inflammation of the aortic valve
b. Progressive stenosis secondary to congenital bicuspid valve
c. Congenital aortic stenosis
d. Idiopathic calcification stenosis of the aortic valve
2. Pathophysiology
Stenosis of the aortic valve results in increased resistance to ventricular
ejection and increased left ventricular pressure. Hypertrophy of the ven-
tricle will occur. Normal aortic valve area is approximately 3 cm2. Aortic valves of <1
cm2 generally produce symptoms, and those with <0.5 cm2 with pressure gradients of
50 mmHg are considered severe.
3. Symptoms
a. Syncope: Commonly with exertion and frequently associated with
vasodilatation in muscle beds, leading to cerebral ischemia.
b. Transient Dysrhythmias
c. Angina
d. CHF
4. Physical Findings
a. Slow-rising, delayed carotid upstroke with decreased amplitude
b. Narrowing of pulse pressure
c. Loud systolic ejection murmur heard at the base of the heart and
radiating to the neck, often with a palpable thrill
5. Diagnostic studies
a. ECG
(1). Left ventricular hypertrophy
(2). Nonspecific repolarization abnormalities
b. Chest x-ray
(1). Pulmonary congestion in patients with CHF
(2). Aortic dilatation
(3). Calcification of the aortic valve
c. Echocardiography
(1). Hypertrophy of the left ventricular wall
(2). Visualization of the abnormal aortic valve
d. Cardiac catheterization documents severity of disease and calculation
of valve area
6. Therapy
a. Judicious management for CHF and angina as they occur (see appro-
priate sections as above. These patients may be very preload sensitive).
b. Valve replacement should be reserved as palliative therapy for patients
who are poor surgical risks.

7.Nursing Diagnosis
a. Activity intolerance related to an imbalance between oxygen supply and demand
as evidenced by exertional dyspnea.
b. Excess fluid volume related to compromised regulatory mechanism as evidenced
by edema, dyspnea, and decreased hematocrit & hemoglobin
c. Decreased cardiac output related to valvular incompetence
8. Desired outcomes
a. The patient with demonstrate increased tolerance to activity by the end of the
shift.
b. The patient will maintain clear lung sounds with no evidence of dyspnea by the
end of the shift.
c. The patient will demonstrate adequate cardiac output as evidenced by blood
pressure and pulse rate and rhythm within normal parameters for client; strong
peripheral pulses; and an ability to tolerate activity without symptoms of dyspnea,
syncope, or chest pain by discharge.

Mitral Stenosis
1. Etiology
a. Rheumatic fever
b. Congenital defects
2. Pathophysiology
The normal mitral orifice is 46 cm2 in area. An obstruction of the orifice results in
impedance of flow into the left ventricle. When the orifice area approaches 1 cm2,
symptoms appear.
3. Symptoms
a. Dyspnea, orthopnea, and paroxysmal nocturnal dyspnea (pulmonary
edema may develop following exertion).
b. Systemic embolization, secondary to thrombi forming in a dilated left
atrium.
c. Dysrhythmias, particularly atrial fibrillation.
d. Hemoptysis, secondary to persistent pulmonary hypertension.
4. Physical Findings
a. Auscultation reveals an opening snap in early diastole.
b. Apical presystolic or mid-diastolic rumble.
c. Accentuated S1, Graham-Steel murmur.
d. Pulmonary regurgitation.
5. Diagnostic Studies
a. ECG: Right ventricle hypertrophy, right axis deviation, left atrial
enlargement, atrial fibrillation
b. Chest X-Ray
(1). Left atrial enlargement is seen on the lateral chest and a double
density on the chest x-ray.
(2). Elevation of the left main stem bronchus and widening of the angle
between the right and left main stem bronchi.
(3). Pulmonary arterial prominence.

c. Echocardiography: Abnormalities of the valve itself may be seen with
calcification and reduction of the E-F slope of the anterior mitral leaflet
during diastole.
6. Treatment
a. Control of ventricular rate in patients with atrial fibrillation and
anticoagulation to prevent thromboembolism.
b. Management of CHF as noted above
7. Nursing Diagnosis
a. Alteration in cardiac output: decreased, due to valvular abnormalities and/or
arrhythmias.
b. Knowledge deficit regarding the nature of the valvular disorder and its
intervention.
c. Knowledge deficit regarding ongoing home self-care.
d. Decreased activity tolerance due to valvular dysfunction and heart failure.
e. Anxiety due to the uncertain nature of the disease and its intervention.
d. Coping deficit due to the chronic nature of the valvular disease and activity
limitations.
8. Desired Outcomes
a. The person will maintain or restore hemodynamic stability, as evidenced by clear
lungs on auscultation, maintenance of stable dry weight, urine output averaging greater
than30 ml per hour, no reported (or observed) dyspnea of orthopnea, normal vital signs,
regular heart rhythm, absence of S3 and 4 heart sounds, and decreased or absent
peripheral edema.
b. The person and/or significant others will demonstrate understanding of the
underlying valvular disorder and prescribed treatment as evidenced by ability to
describe(a) the disease process, (b) factors contributing to symptoms and (c) rationale
for intervention. They will actively participate in the prescribed health behaviors that
enhance success of intervention

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