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Pneumonia Parient Based

This document outlines the pathophysiological process of Pneumocystis pneumonia (PCP) in an 84-year-old heavy smoker. It shows that the predisposing factors of advanced age and smoking weakened the patient's immunity. The contributing factor was infection by the Pneumocystis bacteria, which were able to invade the lungs due to the weakened immunity. This led to progressive lung damage and symptoms of cough, difficulty breathing, and hypoxia as the infection multiplied and caused massive inflammation in the lungs.

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188 views2 pages

Pneumonia Parient Based

This document outlines the pathophysiological process of Pneumocystis pneumonia (PCP) in an 84-year-old heavy smoker. It shows that the predisposing factors of advanced age and smoking weakened the patient's immunity. The contributing factor was infection by the Pneumocystis bacteria, which were able to invade the lungs due to the weakened immunity. This led to progressive lung damage and symptoms of cough, difficulty breathing, and hypoxia as the infection multiplied and caused massive inflammation in the lungs.

Uploaded by

Kismet Summons
Copyright
© Attribution Non-Commercial (BY-NC)
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOC, PDF, TXT or read online on Scribd
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PRECIPITATING FACTORS

PREDISPOSING FACTORS (Modifiable)


(Non-Modifiable)
(+) Heavy Smoker
Age [ 84 y/o ]

CONTRIBUTING FACTOR:
Bacteria- Pneumocystis
Pneumonia (PCP)
“Pneumocystis Jiroveci”

Weakened Progressive Damage to


Immunity Lungs

Entry of Microorganism to
Nasal Passages

Invasion of the
Respiratory System

Activation of
Immune Response Cough
(Mucus Production) [Productive]

Ineffective Immune
Response Results to
Overwhelming Infection

Penetrates the Sterile Lower


Respiratory Tract (Lungs)

Invading lung
Parenchyma

Alveoli Irritation of airway

Multiplies

Colonization Release damaging toxins Increase goblet cell

A B C

A B C
Increase
Occluded the
Infection Mucus
Airway
production
Exudates
Come
Massive Inflammation
From
Inflammation (PNEUMONIA)
Bacteria
Erode the
Lung Vasodilation

Increase Blood
Dead Space Flow
Happened Cough Crackles
Plasma and CHON
Rich Fluid Leakage

Airway
Accumulation of Hyperventilation
Constriction
Edematous Fluid

Inflammed and Fluid


Filled Alveolar Sacs

Alveolar Air Sacs Increased


Become Engorged Respiratory Difficulty of
rate Breathing
(Dyspnea)
Decrease CO2

Impaired O2
Lung consolidation Ventilatory
and CO2
Demands
exchange

Hypoxia

Comprehensive Patient Based Schematic Diagram

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