This document outlines several metabolic cycles and pathways related to oxidative stress and inflammation, including the folate cycle, methionine cycle, urea cycle, arginine pathway, transsulfuration pathway, cobalamin cycle, and neurotransmitter pathways. Key molecules involved include methionine, homocysteine, cysteine, glutathione, dopamine, serotonin, nitric oxide, and ammonia. Imbalances or deficiencies in these cycles and pathways can lead to oxidative damage, microglial activation, and neuronal damage.
This document outlines several metabolic cycles and pathways related to oxidative stress and inflammation, including the folate cycle, methionine cycle, urea cycle, arginine pathway, transsulfuration pathway, cobalamin cycle, and neurotransmitter pathways. Key molecules involved include methionine, homocysteine, cysteine, glutathione, dopamine, serotonin, nitric oxide, and ammonia. Imbalances or deficiencies in these cycles and pathways can lead to oxidative damage, microglial activation, and neuronal damage.
This document outlines several metabolic cycles and pathways related to oxidative stress and inflammation, including the folate cycle, methionine cycle, urea cycle, arginine pathway, transsulfuration pathway, cobalamin cycle, and neurotransmitter pathways. Key molecules involved include methionine, homocysteine, cysteine, glutathione, dopamine, serotonin, nitric oxide, and ammonia. Imbalances or deficiencies in these cycles and pathways can lead to oxidative damage, microglial activation, and neuronal damage.
This document outlines several metabolic cycles and pathways related to oxidative stress and inflammation, including the folate cycle, methionine cycle, urea cycle, arginine pathway, transsulfuration pathway, cobalamin cycle, and neurotransmitter pathways. Key molecules involved include methionine, homocysteine, cysteine, glutathione, dopamine, serotonin, nitric oxide, and ammonia. Imbalances or deficiencies in these cycles and pathways can lead to oxidative damage, microglial activation, and neuronal damage.
