Max-like protein X is a protein that in humans is encoded by the MLX gene.
The product of this gene belongs to the family of basic helix-loop-helix leucine zipper (bHLH-Zip) transcription factors. These factors form heterodimers with Mad proteins and play a role in proliferation, determination and differentiation. This gene product may act to diversify Mad family function by its restricted association with a subset of the Mad family of transcriptional repressors, namely Mad1 and Mad4. Alternatively spliced transcript variants encoding different isoforms have been identified for this gene.
MLX (gene) has been shown to interact with MNT,MXD1 and MLXIPL.
This article incorporates text from the United States National Library of Medicine, which is in the public domain.
MLX or mlx may refer to:
MLX is a series of machine language entry utilities published by COMPUTE! Publications for COMPUTE!, COMPUTE!'s Gazette, and various books. These programs were designed to allow relatively easy entry of the type-in machine language listings that were often included in these publications. Versions were available for the Commodore 64, PET and VIC 20, the Atari 8-bit family and the Apple II series.
MLX was first introduced in the December 1983 issue of Compute!, with versions for both the Commodore 64 and Atari 8-bit family. This was followed the next month by a version for the VIC-20 ("Tiny MLX") that was otherwise similar.
These versions of MLX shared a format consisting of six data bytes in decimal format, plus a seventh byte which served as a checksum. Although each line began with the memory address, and bytes were separated with commas, these did not have to be typed; the program auto-incremented the address and automatically printed the comma delimiters every three characters. Invalid keystrokes were filtered out. When a line was completely typed, it was automatically verified against the checksum value. If it matched, the user would be prompted for the next line. If not, a warning buzzer would sound and the invalid input would be discarded so that the user could retype the line. Several keyboard keys were redefined to create a makeshift numeric keypad.
The SCMaglev (superconducting maglev) (formerly called the MLU) is a magnetic levitation (maglev) railway system based on the principle of magnetic repulsion between the track and the cars. The Central Japan Railway Company (JR Central) and the company's Railway Technical Research Institute developed the system.
On 21 April 2015, a manned seven-car L0 series SCMaglev train reached a speed of 603 km/h (375 mph), less than a week after the same train clocked 590 km/h (370 mph), breaking the previous land speed record for rail vehicles of 581 km/h (361 mph) set by a JR Central MLX01 maglev train in December 2003.
The SCMaglev system uses an electrodynamic suspension (EDS) system. Installed in the trains' bogies are superconducting magnets, and the guideways contain two sets of metal coils.
The current levitation system utilizes a series of coils wound into a "figure 8" along both walls of the guideway. These coils are also cross-connected underneath the track.
As the train accelerates, the magnetic fields of its superconducting magnets induce a current into these coils due to the magnetic field induction effect. If the train were centered with the coils, the electrical potential would be balanced and no currents would be induced. However, as the train runs on rubber wheels at relatively low speeds, the magnetic fields are positioned below the center of the coils, causing the electrical potential to no longer be balanced. This creates a reactive magnetic field opposing the superconducting magnet's pole (in accordance with Lenz's law), and a pole above that attracts it. Once the train reaches 150 km/h (93 mph), there is sufficient current flowing to lift the train 100 mm (4 in) above the guideway.
A gene is a locus (or region) of DNA that encodes a functional RNA or protein product, and is the molecular unit of heredity. The transmission of genes to an organism's offspring is the basis of the inheritance of phenotypic traits. Most biological traits are under the influence of polygenes (many different genes) as well as the gene–environment interactions. Some genetic traits are instantly visible, such as eye colour or number of limbs, and some are not, such as blood type, risk for specific diseases, or the thousands of basic biochemical processes that comprise life.
Genes can acquire mutations in their sequence, leading to different variants, known as alleles, in the population. These alleles encode slightly different versions of a protein, which cause different phenotype traits. Colloquial usage of the term "having a gene" (e.g., "good genes," "hair colour gene") typically refers to having a different allele of the gene. Genes evolve due to natural selection or survival of the fittest of the alleles.
Eugene is a common (masculine) first name that comes from the Greek εὐγενής (eugenēs), "noble", literally "well-born", from εὖ (eu), "well" and γένος (genos), "race, stock, kin".Gene is a common shortened form. The feminine variant is Eugenia or Eugénie.
Male foreign-language variants include:
Gene is a thriller novel by Stel Pavlou (born 1970), published in 2005 in England by Simon & Schuster. It is published in several languages with some title changes. The Italian edition has the title La Conspirazione del Minotauro (The Minotaur Conspiracy). The novel is about a fictional New York detective, James North, who in the process of hunting down a criminal, uncovers a genetics experiment to unlock past lives through genetic memory, therefore achieving a kind of immortality. In so doing North discovers his own origins, that of a soldier from the Trojan War who is reincarnated seven times through history, forced to confront his nemesis each time, all for the loss of his one true love.
Cyclades (born circa 1300 BC)
Incarnations of Cyclades
Athanatos (born circa 1500 BC)
Incarnations of Athanatos