Leda and similar may refer to:
Leda (/ˈliːdə/ LEE-də; Greek: Λήδα), also known as Jupiter XIII, is a prograde irregular satellite of Jupiter. It was discovered by Charles T. Kowal at the Mount Palomar Observatory on September 14, 1974, after three nights' worth of photographic plates had been taken (September 11 through 13; Leda appears on all of them). It was named after Leda, who was a lover of Zeus, the Greek equivalent of Jupiter (who came to her in the form of a swan). Kowal suggested the name and the IAU endorsed it in 1975.
Leda belongs to the Himalia group, five moons orbiting between 11 and 13 Gm from Jupiter at an inclination of about 27.5°. The orbital elements given here are as of January 2000, but they are continuously changing due to solar and planetary perturbations.
Web of Passion (also released as Leda, original French title: À double tour) is a 1959 French suspense thriller film directed by Claude Chabrol and based on the novel The Key to Nicholas Street by American writer Stanley Ellin. It was Chabrol's first film in the thriller genre, which would be his genre of choice for the rest of his career. The film had a total of 1,445,587 admissions in France.
Leda (Antonella Lualdi), the mistress of the wealthy Henri Marcoux is murdered and the family accuses the milkman of committing the crime. But Marcoux's daughter's fiance (Jean-Paul Belmondo) suspects that Leda may have been murdered by someone else.
Belmondo plays a character named Laszlo Kovacs, which was the alias of his character Michel Poiccard in Breathless
GM1 (monosialotetrahexosylganglioside) the "prototype" ganglioside, is a member of the ganglio series of gangliosides which contain one sialic acid residue. GM1 has important physiological properties and impacts neuronal plasticity and repair mechanisms, and the release of neurotrophins in the brain. Besides its function in the physiology of the brain, GM1 acts as the site of binding for both Cholera toxin and E. coli heat-labile enterotoxin (Traveller's diarrhea).
Galactosidases are enzymes that breakdown GM1, and the failure to remove GM1 results in GM1 gangliosidosis. GM1 gangliosidosis are inherited disorders that progressively destroys neurons in the brain and spinal cord as GM1 accumulates. Without treatment, this results in developmental decline and muscle weakness, eventually leading to severe retardation and death.
Antibodies to GM1 are increased in Guillain-Barré syndrome, dementia and lupus but their function is not clear. There is some evidence to suggest antibodies against GM1 are associated with diarrhea in Guillain-Barré syndrome.