Korean Central News Agency

The Korean Central News Agency (KCNA) is the state news agency of North Korea that was established on December 5, 1946. The agency portrays the views of the Workers' Party of Korea and the North Korean government for foreign consumption. KCNA is headquartered in the capital city of Pyongyang. In South Korea, access to both KCNA websites: www.kcna.kp and www.kcna.co.jp, is blocked by the South Korean government.

Organization

As the sole news agency of North Korea, KCNA daily reports news for all the North Korean news organizations including newspapers, radio and television broadcasts via Korean Central Television and the Korean Central Broadcasting System within the country. In December 1996, KCNA began publishing its news articles on the Internet with its web server located in Japan. Since October 2010, stories have been published on a new site, controlled from Pyongyang, and output has been significantly increased to include world stories with no specific link to North Korea as well as news from countries that have strong DPRK ties.

KCNA-FM

KCNA (102.7 FM, "102.7 The Drive") is a commercial classic hits music radio station in Cave Junction, Oregon, broadcasting to the Medford-Ashland, Oregon area. The station is currently owned by Opus Broadcasting Systems.

Translators and booster

KCNA broadcasts on the following translators and booster:

References

External links

KCNA5

Potassium voltage-gated channel, shaker-related subfamily, member 5, also known as KCNA5 or K_v1.5, is a protein that in humans is encoded by the KCNA5 gene.

Function

Potassium channels represent the most complex class of voltage-gated ion channels from both functional and structural standpoints. KCNA5 encodes a member of the potassium channel, voltage-gated, shaker-related subfamily. This member contains six membrane-spanning domains with a shaker-type repeat in the fourth segment. It belongs to the delayed rectifier class, the function of which could restore the resting membrane potential of beta cells after depolarization, thereby contributing to the regulation of insulin secretion. This gene is intronless, and the gene is clustered with genes KCNA1 and KCNA6 on chromosome 12. Mutations in this gene have been related to both atrial fibrillation and sudden cardiac death. KCNA5 are also key players in pulmonary vascular function, where they play a role in setting the resting membrane potential and its involvement during hypoxic pulmonary vasoconstriction.