Éber Alejandro Moas Silveira (born March 21, 1969 in Montevideo) is a retired football defender from Uruguay, who obtained 48 international caps for his national team. Having made his debut on September 27, 1988 against Ecuador (2-1), Moas played his last international match for his native country on July 20, 1997 in a World Cup Qualifier against Bolivia (0-1).
Moas started his playing career in 1988 with Danubio. He joined Club Atlético Independiente of Argentina in 1992 where he played 65 games.
In 1995 he joined América de Cali of Colombia and in 1996 he went to Mexico to play for Monterrey.
In 1998 Moas returned to Danubio where he played until 2002. In 2005 he joined Racing Club de Montevideo and in 2007 he played for Rentistas.
Eber (עֵבֶר, ISO 259-3 ʕebr, Standard Hebrew Éver, Tiberian Hebrew ʻĒḇer) is an ancestor of the Israelites, according to the "Table of Nations" in Genesis 10-11 and 1 Chronicles 1. He was a great-grandson of Noah's son Shem and the father of Peleg born when Eber was 34 years old, and of Joktan. He was the son of Shelah a distant ancestor of Abraham. According to the Hebrew Bible, Shelah died at the age of 464 (Genesis 11:14-17) when Jacob was 20. The Hebrew Calendar synchronises this date with 1817 BCE, however numerous other dates have been proposed for the patriarchs in Genesis.
In the Septuagint and other Christian Bibles derived from it, Eber is called Heber and his father is called Sala. His son is called Phaleg, born when Heber was 134 years old, and he had other sons and daughters. Heber lived to an age of 404 years. (Septuagint Genesis 11:14-17)
In Jewish tradition, Eber, the great-grandson of Shem, refused to help with the building of the Tower of Babel, so his language was not confused when it was abandoned. He and his family alone retained the original human language, Hebrew, a language named after Eber (Heber), also called lingua humana in Latin. (There are different religious positions on this issue; see also Adamic language.)
The Epstein–Barr virus-encoded small RNAs (EBERs) are small non-coding RNAs localized in the nucleus of human cells infected with Epstein–Barr virus (EBV). First discovered in 1981, EBERs are the most abundant RNAs present in infected cells. EBERs interact with several host proteins to form ribonucleoprotein (RNP) complexes. Although a precise function for EBERs remains elusive, roles in transformation and oncogenesis are proposed.
EBER1 and EBER2 are short, 167 and 172 nucleotides in length respectively, nuclear-enriched non-coding RNAs. These two RNAs are transcribed by the host's RNA polymerase III during latent infection of EBV.
EBERs 1 and 2 can be deleted from the viral genome without noticeable phenotypic changes, though this has never been found deleted in nature. EBER expression alone can induce tumours in severe combined immunodificient mice.
EBER1 associates with human ribosomal protein L22 and causes this protein to move from the nucleolus to the nucleoplasm. EBER1 also specifically interacts with at least three host hnRNPs (A1, A2/B1 and D/AUF1).