Pages that link to "Q78762648"
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The following pages link to CD8 alpha beta T cells are not essential to the pathogenesis of arthritis or colitis in HLA-B27 transgenic rats (Q78762648):
Displayed 50 items.
- HLA-B27 and psoriatic disease: a modern view of an old relationship (Q26796433) (← links)
- Review: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyond (Q26991709) (← links)
- HLA-B27-Homodimer-Specific Antibody Modulates the Expansion of Pro-Inflammatory T-Cells in HLA-B27 Transgenic Rats (Q27303777) (← links)
- HLA-B27 and human β2-microglobulin affect the gut microbiota of transgenic rats (Q27313830) (← links)
- From HLA-B27 to spondyloarthritis: a journey through the ER. (Q27687662) (← links)
- CD8 alpha is expressed by human monocytes and enhances Fc gamma R-dependent responses. (Q33293227) (← links)
- Spondyloarthritis: Matrix Metalloproteinasesas Biomarkers of Pathogenesis and Response to Tumor Necrosis Factor (TNF) Inhibitors (Q33624763) (← links)
- Overexpression and unique rearrangement of VH2 transcripts in immunoglobulin variable heavy chain genes in ankylosing spondylitis patients (Q33879851) (← links)
- Endoplasmic reticulum aminopeptidase-1 alleles associated with increased risk of ankylosing spondylitis reduce HLA-B27 mediated presentation of multiple antigens. (Q33924181) (← links)
- HLA-B27 misfolding and the unfolded protein response augment interleukin-23 production and are associated with Th17 activation in transgenic rats (Q33945978) (← links)
- An overview of genetics of paediatric rheumatic diseases (Q34363289) (← links)
- Classification of juvenile spondyloarthritis: Enthesitis-related arthritis and beyond (Q34363306) (← links)
- Reverse interferon signature is characteristic of antigen-presenting cells in human and rat spondyloarthritis. (Q34481463) (← links)
- Pathogenicity of Misfolded and Dimeric HLA-B27 Molecules (Q34921584) (← links)
- Cytokines and chemokines in uveitis: is there a correlation with clinical phenotype? (Q35575081) (← links)
- Extraintestinal complications of inflammatory bowel disease (Q35577416) (← links)
- HLA-B27???associated uveitis: overview and current perspectives (Q35582795) (← links)
- Ankylosing spondylitis macrophage production of higher levels of interleukin-23 in response to lipopolysaccharide without induction of a significant unfolded protein response (Q35585432) (← links)
- In vivo pre-activation of monocytes in patients with axial spondyloarthritis (Q35856805) (← links)
- Ankylosing spondylitis patients display altered dendritic cell and T cell populations that implicate pathogenic roles for the IL-23 cytokine axis and intestinal inflammation (Q36366157) (← links)
- Sulfasalazine Treatment Suppresses the Formation of HLA-B27 Heavy Chain Homodimer in Patients with Ankylosing Spondylitis (Q36512479) (← links)
- Pathogenesis of spondyloarthritis: insights from synovial membrane studies (Q36535213) (← links)
- Silencing or inhibition of endoplasmic reticulum aminopeptidase 1 (ERAP1) suppresses free heavy chain expression and Th17 responses in ankylosing spondylitis. (Q36859633) (← links)
- The unfolded protein response and gastrointestinal disease (Q36866561) (← links)
- HLA-B27 misfolding and spondyloarthropathies (Q37178385) (← links)
- Pathogenesis of ankylosing spondylitis. (Q37762157) (← links)
- Novel HLA-B27-restricted epitopes from Chlamydia trachomatis generated upon endogenous processing of bacterial proteins suggest a role of molecular mimicry in reactive arthritis (Q37846210) (← links)
- Spondyloarthritis (Q37891477) (← links)
- IL-23 responsive innate-like T cells in spondyloarthritis: the less frequent they are, the more vital they appear. (Q38421287) (← links)
- Revisiting MHC genes in spondyloarthritis (Q38436508) (← links)
- Innate Immune Activation Can Trigger Experimental Spondyloarthritis in HLA-B27/Huβ2m Transgenic Rats (Q38616684) (← links)
- The role of the unfolded protein response in axial spondyloarthritis (Q38633365) (← links)
- Autoinflammation and HLA-B27: Beyond Antigen Presentation (Q38845301) (← links)
- Peptide handling by HLA-B27 subtypes influences their biological behavior, association with ankylosing spondylitis and susceptibility to endoplasmic reticulum aminopeptidase 1 (ERAP1). (Q38959953) (← links)
- Induction of HLA-B27 heavy chain homodimer formation after activation in dendritic cells (Q39945166) (← links)
- Major histocompatibility complex class I-restricted alloreactive CD4+ T cells. (Q40564855) (← links)
- Recombinant Salmonella typhimurium outer membrane protein A is recognized by synovial fluid CD8 cells and stimulates synovial fluid mononuclear cells to produce interleukin (IL)-17/IL-23 in patients with reactive arthritis and undifferentiated spond (Q40722205) (← links)
- Expression of HLA-B27 causes loss of migratory dendritic cells in a rat model of spondylarthritis (Q41859496) (← links)
- Suppression of Inflammation and Arthritis by Orally Administrated Cardiotoxin from Naja naja atra (Q43164426) (← links)
- IL-23 induces spondyloarthropathy by acting on ROR-γt+ CD3+CD4-CD8- entheseal resident T cells (Q46043893) (← links)
- Adoptive transfer of nontransgenic mesenteric lymph node cells induces colitis in athymic HLA-B27 transgenic nude rats (Q46951072) (← links)
- Defective costimulatory function is a striking feature of antigen-presenting cells in an HLA-B27-transgenic rat model of spondylarthropathy (Q47596771) (← links)
- Reduced ratio of protective versus proinflammatory cytokine responses to commensal bacteria in HLA-B27 transgenic rats (Q47927316) (← links)
- Association between ED in ankylosing spondylitis: a population-based study. (Q48185593) (← links)
- Effects of HLA-B27 on Gut Microbiota in Experimental Spondyloarthritis Implicate an Ecological Model of Dysbiosis (Q49491551) (← links)
- Autoreactivity to self H-2Kb peptides in TAP1 mice. Intravenous administration of H-2Kb class I-derived peptides induces long-term survival of grafts from C57BL/6 donors. (Q53666747) (← links)
- Regulation of Cytokine Production by the Unfolded Protein Response; Implications for Infection and Autoimmunity. (Q55260956) (← links)
- Whodunit? The Contribution of Interleukin (IL)-17/IL-22-Producing γδ T Cells, αβ T Cells, and Innate Lymphoid Cells to the Pathogenesis of Spondyloarthritis. (Q55339808) (← links)
- Primed for inflammation: enthesis-resident T cells (Q57231913) (← links)
- Disease-associated polymorphisms in ERAP1 do not alter endoplasmic reticulum stress in patients with ankylosing spondylitis (Q57667207) (← links)