Pages that link to "Q52169941"
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The following pages link to Conditional mutagenesis in mice with heat shock promoter-driven cre transgenes. (Q52169941):
Displaying 50 items.
- Placental overgrowth in mice lacking the imprinted gene Ipl (Q24530568) (← links)
- Conditional knockout mice reveal distinct functions for the global transcriptional coactivators CBP and p300 in T-cell development (Q24537650) (← links)
- Role of IGF-I signaling in muscle bone interactions (Q28082188) (← links)
- Insulin-like growth factor (IGF) signaling through type 1 IGF receptor plays an important role in remyelination (Q28584984) (← links)
- Mutation of the murine Bloom's syndrome gene produces global genome destabilization (Q28585807) (← links)
- Insulin and IGF1 receptors are essential for XX and XY gonadal differentiation and adrenal development in mice (Q28589118) (← links)
- Genetic ablation reveals that the roof plate is essential for dorsal interneuron specification (Q28594999) (← links)
- Macrophages redirect phagocytosis by non-professional phagocytes and influence inflammation (Q30275465) (← links)
- TASK channels determine pH sensitivity in select respiratory neurons but do not contribute to central respiratory chemosensitivity. (Q30445251) (← links)
- Subregion-specific p300 conditional knock-out mice exhibit long-term memory impairments (Q30470054) (← links)
- An essential role for insulin and IGF1 receptors in regulating sertoli cell proliferation, testis size, and FSH action in mice (Q33635803) (← links)
- Insulin-like growth factor-1 induces regulatory T cell-mediated suppression of allergic contact dermatitis in mice (Q33935628) (← links)
- Two nested developmental waves demarcate a compartment boundary in the mouse lung (Q33969488) (← links)
- Deletion of exon 20 of the Familial Dysautonomia gene Ikbkap in mice causes developmental delay, cardiovascular defects, and early embryonic lethality (Q34064901) (← links)
- Distinct growth hormone receptor signaling modes regulate skeletal muscle development and insulin sensitivity in mice. (Q34245339) (← links)
- Flexible Accelerated STOP Tetracycline Operator-knockin (FAST): a versatile and efficient new gene modulating system (Q34263796) (← links)
- A method for the generation of conditional gene repair mutations in mice (Q34582775) (← links)
- Rapid generation of inducible mouse mutants (Q34771977) (← links)
- Gene therapy for cardiac cachexia? (Q34774475) (← links)
- Recent advances in inducible expression in transgenic mice (Q34870765) (← links)
- Myc is a Notch1 transcriptional target and a requisite for Notch1-induced mammary tumorigenesis in mice (Q35039989) (← links)
- Heat shock modulates neutrophil motility in zebrafish (Q35075761) (← links)
- A mouse model of uterine leiomyosarcoma (Q35083288) (← links)
- Osteoblast-restricted Disruption of the Growth Hormone Receptor in Mice Results in Sexually Dimorphic Skeletal Phenotypes (Q35755050) (← links)
- Transgenic rescue of insulin receptor-deficient mice (Q35835529) (← links)
- Heat shock-inducible Cre/Lox approaches to induce diverse types of tumors and hyperplasia in transgenic zebrafish (Q35839623) (← links)
- Fibroblast growth factor receptor 2 regulates proliferation and Sertoli differentiation during male sex determination (Q36082256) (← links)
- IKAP expression levels modulate disease severity in a mouse model of familial dysautonomia (Q36375822) (← links)
- IGF-1R signaling in chondrocytes modulates growth plate development by interacting with the PTHrP/Ihh pathway (Q36488507) (← links)
- The basal-like mammary carcinomas induced by Brca1 or Bard1 inactivation implicate the BRCA1/BARD1 heterodimer in tumor suppression (Q36627600) (← links)
- Regulation of Ligand and Shear Stress-induced Insulin-like Growth Factor 1 (IGF1) Signaling by the Integrin Pathway. (Q36778833) (← links)
- Notch signaling maintains Leydig progenitor cells in the mouse testis (Q36842267) (← links)
- The hormonal action of IGF1 in postnatal mouse growth (Q37039560) (← links)
- Insulin-like growth factor-1 receptor in mature osteoblasts is required for periosteal bone formation induced by reloading (Q37105615) (← links)
- Igf1r as a therapeutic target in a mouse model of basal-like breast cancer (Q37114859) (← links)
- Mouse chimeras and their application to circadian biology (Q37222648) (← links)
- Maternal imprinting at the H19-Igf2 locus maintains adult haematopoietic stem cell quiescence. (Q37497577) (← links)
- Good planning and serendipity: exploiting the Cre/Lox system in the testis (Q37810390) (← links)
- Ephrin B2/EphB4 mediates the actions of IGF-I signaling in regulating endochondral bone formation (Q38308154) (← links)
- Autocrine and Paracrine Actions of IGF-I Signaling in Skeletal Development (Q38566647) (← links)
- IGF1-Dependent Synaptic Plasticity of Mitral Cells in Olfactory Memory during Social Learning (Q38690906) (← links)
- Pathophysiology of the Desmo-Adhesome (Q38922797) (← links)
- 5-HT1A autoreceptor levels determine vulnerability to stress and response to antidepressants (Q39281683) (← links)
- Defective insulin secretion in pancreatic beta cells lacking type 1 IGF receptor (Q39738488) (← links)
- Mosaic analysis of insulin receptor function (Q40413498) (← links)
- Phosphorylation of Human Rad9 Is Required for Genotoxin-activated Checkpoint Signaling (Q40653014) (← links)
- IGF-I Signaling in Osterix-Expressing Cells Regulates Secondary Ossification Center Formation, Growth Plate Maturation, and Metaphyseal Formation During Postnatal Bone Development (Q40901506) (← links)
- Osteoblast-Specific Loss of IGF1R Signaling Results in Impaired Endochondral Bone Formation During Fracture Healing (Q41174769) (← links)
- Deletion of mouse rad9 causes abnormal cellular responses to DNA damage, genomic instability, and embryonic lethality (Q41283109) (← links)
- Differential organ phenotypes after postnatal Igf1r gene conditional deletion induced by tamoxifen in UBC-CreERT2; Igf1r fl/fl double transgenic mice. (Q41929403) (← links)