Pages that link to "Q42435626"

The following pages link to Effects of barium and ion substitutions in artificial blood on endocochlear potential (Q42435626):

Displaying 36 items.

• Inwardly rectifying potassium channels: their structure, function, and physiological roles (Q24296441) (← links)
• The endocochlear potential depends on two K+ diffusion potentials and an electrical barrier in the stria vascularis of the inner ear (Q24652355) (← links)
• SLC26A4 targeted to the endolymphatic sac rescues hearing and balance in Slc26a4 mutant mice. (Q27321123) (← links)
• The unique electrical properties in an extracellular fluid of the mammalian cochlea; their functional roles, homeostatic processes, and pathological significance. (Q28076301) (← links)
• K+ cycling and the endocochlear potential (Q28198160) (← links)
• Slc26a4-insufficiency causes fluctuating hearing loss and stria vascularis dysfunction (Q30410497) (← links)
• The role of an inwardly rectifying K(+) channel (Kir4.1) in the inner ear and hearing loss (Q30414096) (← links)
• ILDR1 null mice, a model of human deafness DFNB42, show structural aberrations of tricellular tight junctions and degeneration of auditory hair cells (Q30420205) (← links)
• Loss of cochlear HCO3- secretion causes deafness via endolymphatic acidification and inhibition of Ca2+ reabsorption in a Pendred syndrome mouse model (Q30494661) (← links)
• Three-dimensional and ultrastructural relationships between intermediate cells and capillaries in the gerbil stria vascularis (Q30665973) (← links)
• Epithelial cell stretching and luminal acidification lead to a retarded development of stria vascularis and deafness in mice lacking pendrin (Q33851694) (← links)
• Cochlear blood flow following temporary occlusion of the cerebellar arteries (Q34768103) (← links)
• Mouse model of enlarged vestibular aqueducts defines temporal requirement of Slc26a4 expression for hearing acquisition (Q35485037) (← links)
• Supporting sensory transduction: cochlear fluid homeostasis and the endocochlear potential (Q36542382) (← links)
• Molecular and physiological bases of the K+ circulation in the mammalian inner ear. (Q36600743) (← links)
• Stria vascularis and vestibular dark cells: characterisation of main structures responsible for inner-ear homeostasis, and their pathophysiological relations (Q37197118) (← links)
• How is the highly positive endocochlear potential formed? The specific architecture of the stria vascularis and the roles of the ion-transport apparatus (Q37655404) (← links)
• Mechanism generating endocochlear potential: role played by intermediate cells in stria vascularis (Q40173673) (← links)
• Computer modeling defines the system driving a constant current crucial for homeostasis in the mammalian cochlea by integrating unique ion transports. (Q41527578) (← links)
• Ion transport mechanisms responsible for K+ secretion and the transepithelial voltage across marginal cells of stria vascularis in vitro (Q41679999) (← links)
• Hearing Loss Controlled by Optogenetic Stimulation of Nonexcitable Nonglial Cells in the Cochlea of the Inner Ear (Q42172817) (← links)
• An ATP-dependent inwardly rectifying potassium channel, KAB-2 (Kir4. 1), in cochlear stria vascularis of inner ear: its specific subcellular localization and correlation with the formation of endocochlear potential. (Q42439036) (← links)
• KCNJ10 (Kir4.1) potassium channel knockout abolishes endocochlear potential (Q42515610) (← links)
• Reduction in the endocochlear potential caused by Cs(+) in the perilymph can be explained by the five-compartment model of the stria vascularis (Q42522324) (← links)
• The change in the electrical resistance of the scala media produced by vasopressin (Q42523995) (← links)
• Transepithelial electrical potential of nonsensory region of gerbil utricle in vitro (Q44374073) (← links)
• Changes in immunostaining of cochleas with experimentally induced endolymphatic hydrops (Q46143354) (← links)
• Immunolocalization of Na+, K(+)-ATPase, Ca(++)-ATPase, calcium-binding proteins, and carbonic anhydrase in the guinea pig inner ear. (Q46730722) (← links)
• Effects of forskolin and 1,9-dideoxy-forskolin on cochlear potentials (Q46773260) (← links)
• Intravascularly applied K(+)-channel blockers suppress differently the positive endocochlear potential maintained by vascular perfusion (Q46895050) (← links)
• Inwardly rectifying K+ currents in intermediate cells in the cochlea of gerbils: a possible contribution to the endocochlear potential (Q47876563) (← links)
• The anterior inferior cerebellar arterial network supplying the rat cochlea and its role in autoregulation of cochlear blood flow (Q48585244) (← links)
• Gastric type H+,K+-ATPase in the cochlear lateral wall is critically involved in formation of the endocochlear potential (Q50463548) (← links)
• The mechanism underlying maintenance of the endocochlear potential by the K+ transport system in fibrocytes of the inner ear. (Q50850272) (← links)
• Morphological Aspects of Transport of Potassium Ion in the Marginal Cell (Q58081887) (← links)
• Two types of K+ currents in marginal cells cultured from rat stria vascularis (Q73875702) (← links)