Pages that link to "Q38326803"
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The following pages link to TDP-43 and FUS RNA-binding proteins bind distinct sets of cytoplasmic messenger RNAs and differently regulate their post-transcriptional fate in motoneuron-like cells. (Q38326803):
Displayed 50 items.
- Mechanisms of FUS mutations in familial amyotrophic lateral sclerosis (Q26749169) (← links)
- The extreme N-terminus of TDP-43 mediates the cytoplasmic aggregation of TDP-43 and associated toxicity in vivo (Q26751400) (← links)
- Dysregulated axonal RNA translation in amyotrophic lateral sclerosis (Q26753110) (← links)
- The Function of Autophagy in Neurodegenerative Diseases (Q26777018) (← links)
- Converging mechanisms in ALS and FTD: disrupted RNA and protein homeostasis (Q26863439) (← links)
- The function of RNA-binding proteins at the synapse: implications for neurodegeneration (Q26991684) (← links)
- RNA-binding proteins in neurodegeneration: Seq and you shall receive (Q27000416) (← links)
- A loss of FUS/TLS function leads to impaired cellular proliferation (Q27331026) (← links)
- Early lethality and neuronal proteinopathy in mice expressing cytoplasm-targeted FUS that lacks the RNA recognition motif. (Q27334411) (← links)
- The crystal structure of TDP-43 RRM1-DNA complex reveals the specific recognition for UG- and TG-rich nucleic acids (Q27681463) (← links)
- EWS and FUS bind a subset of transcribed genes encoding proteins enriched in RNA regulatory functions (Q28607398) (← links)
- A deep learning framework for modeling structural features of RNA-binding protein targets (Q30390089) (← links)
- Biochemical Properties and Biological Functions of FET Proteins (Q30459373) (← links)
- Axonal transport of TDP-43 mRNA granules is impaired by ALS-causing mutations (Q30573122) (← links)
- FUS/TLS assembles into stress granules and is a prosurvival factor during hyperosmolar stress (Q30577079) (← links)
- Astrocytic TDP-43 pathology in Alexander disease (Q33575134) (← links)
- TDP-43 suppresses tau expression via promoting its mRNA instability (Q33740999) (← links)
- GraphProt: modeling binding preferences of RNA-binding proteins. (Q33741982) (← links)
- Functions of FUS/TLS from DNA repair to stress response: implications for ALS. (Q34305789) (← links)
- The expression levels of microRNA-361-5p and its target VEGFA are inversely correlated in human cutaneous squamous cell carcinoma (Q34482531) (← links)
- Pur-alpha regulates cytoplasmic stress granule dynamics and ameliorates FUS toxicity. (Q34507318) (← links)
- Targeting RNA binding proteins involved in neurodegeneration (Q34954162) (← links)
- ALS-associated FUS mutations result in compromised FUS alternative splicing and autoregulation (Q35034350) (← links)
- ALS-causative mutations in FUS/TLS confer gain and loss of function by altered association with SMN and U1-snRNP. (Q35114564) (← links)
- A network of RNA and protein interactions in Fronto Temporal Dementia (Q35194634) (← links)
- Linking RNA Dysfunction and Neurodegeneration in Amyotrophic Lateral Sclerosis (Q35501802) (← links)
- FUS/TLS contributes to replication-dependent histone gene expression by interaction with U7 snRNPs and histone-specific transcription factors (Q35739332) (← links)
- Altered mRNP granule dynamics in FTLD pathogenesis. (Q35944221) (← links)
- Progranulin: a proteolytically processed protein at the crossroads of inflammation and neurodegeneration (Q36294670) (← links)
- A combined sequence and structure based method for discovering enriched motifs in RNA from in vivo binding data (Q36301366) (← links)
- TDP-43 and FUS en route from the nucleus to the cytoplasm (Q36336278) (← links)
- A deep boosting based approach for capturing the sequence binding preferences of RNA-binding proteins from high-throughput CLIP-seq data (Q36391211) (← links)
- Long non-coding RNA gadd7 interacts with TDP-43 and regulates Cdk6 mRNA decay (Q36440463) (← links)
- RNA-binding ability of FUS regulates neurodegeneration, cytoplasmic mislocalization and incorporation into stress granules associated with FUS carrying ALS-linked mutations (Q36626888) (← links)
- Divergent roles of ALS-linked proteins FUS/TLS and TDP-43 intersect in processing long pre-mRNAs (Q36650975) (← links)
- Stress granules as crucibles of ALS pathogenesis. (Q36804082) (← links)
- RNA in unexpected places: long non-coding RNA functions in diverse cellular contexts (Q36855920) (← links)
- Amyotrophic lateral sclerosis-linked FUS/TLS alters stress granule assembly and dynamics (Q37153932) (← links)
- ALS-associated mutant FUS inhibits macroautophagy which is restored by overexpression of Rab1 (Q37164240) (← links)
- RNA-mediated toxicity in neurodegenerative disease (Q37216891) (← links)
- The ALS/FTLD-related RNA-binding proteins TDP-43 and FUS have common downstream RNA targets in cortical neurons (Q37364300) (← links)
- Tat-activating regulatory DNA-binding protein regulates glycolysis in hepatocellular carcinoma by regulating the platelet isoform of phosphofructokinase through microRNA 520. (Q37582377) (← links)
- Structural disorder and the loss of RNA homeostasis in aging and neurodegenerative disease. (Q38130678) (← links)
- FUS-mediated alternative splicing in the nervous system: consequences for ALS and FTLD. (Q38131381) (← links)
- Why is ALS so Difficult to Treat? (Q38188662) (← links)
- RNA metabolism in ALS: when normal processes become pathological. (Q38189807) (← links)
- RNA-binding proteins in neurological diseases. (Q38198537) (← links)
- Pathogenesis/genetics of frontotemporal dementia and how it relates to ALS. (Q38219206) (← links)
- High throughput platform to explore RNA-protein interactomes (Q38229626) (← links)
- Microarray gene and miRNA expression studies: looking for new therapeutic targets for frontotemporal lobar degeneration. (Q38246624) (← links)