Pages that link to "Q33963311"
Jump to navigation
Jump to search
The following pages link to Inactivation of p53 by human T-cell lymphotropic virus type 1 Tax requires activation of the NF-kappaB pathway and is dependent on p53 phosphorylation. (Q33963311):
Displayed 50 items.
- Hostile takeovers: viral appropriation of the NF-kB pathway (Q22241900) (← links)
- NF-kappaB prevents cells from undergoing Cr(VI)-induced apoptosis (Q23922085) (← links)
- The functional genomic response of developing embryonic submandibular glands to NF-kappa B inhibition (Q24794871) (← links)
- Human T-cell leukemia virus type I (HTLV-I) infection and the onset of adult T-cell leukemia (ATL) (Q24805664) (← links)
- The role of cyclin D2 and p21/waf1 in human T-cell leukemia virus type 1 infected cells (Q24807093) (← links)
- New insights into the role of nuclear factor-kB in cell growth regulation (Q26058119) (← links)
- Modes of Human T Cell Leukemia Virus Type 1 Transmission, Replication and Persistence (Q26801294) (← links)
- Modulation of DNA damage and repair pathways by human tumour viruses (Q26825783) (← links)
- Tumor Suppressor Inactivation in the Pathogenesis of Adult T-Cell Leukemia (Q28081287) (← links)
- Effect of transforming viruses on molecular mechanisms associated with cancer (Q28273926) (← links)
- Tax-1 and Tax-2 similarities and differences: focus on post-translational modifications and NF-κB activation. (Q30353061) (← links)
- Human T-cell leukemia virus I tax protein sensitizes p53-mutant cells to DNA damage (Q30446360) (← links)
- Complex cell cycle abnormalities caused by human T-lymphotropic virus type 1 Tax. (Q30499255) (← links)
- An NF-kappaB and slug regulatory loop active in early vertebrate mesoderm (Q33268281) (← links)
- Combination of 9-aminoacridine with Campath-1H provides effective therapy for a murine model of adult T-cell leukemia (Q33767881) (← links)
- Differences in the ability of human T-cell lymphotropic virus type 1 (HTLV-1) and HTLV-2 tax to inhibit p53 function (Q33808681) (← links)
- Genetic evidence of a role for ATM in functional interaction between human T-cell leukemia virus type 1 Tax and p53. (Q33834966) (← links)
- Evidence for NF-kappaB- and CBP-independent repression of p53's transcriptional activity by human T-cell leukemia virus type 1 Tax in mouse embryo and primary human fibroblasts (Q33884079) (← links)
- Conditional ablation of Ikkb inhibits melanoma tumor development in mice (Q33968131) (← links)
- The Rel/NF-kappa B family: friend and foe. (Q34023268) (← links)
- Viral replication and the coactivators p300 and CBP. (Q34104253) (← links)
- Seizing of T cells by human T-cell leukemia/lymphoma virus type 1. (Q34273466) (← links)
- Physical interaction of human T-cell leukemia virus type 1 Tax with cyclin-dependent kinase 4 stimulates the phosphorylation of retinoblastoma protein (Q34278817) (← links)
- Molecular mechanisms of cellular transformation by HTLV-1 Tax. (Q34450092) (← links)
- Fuse binding protein antagonizes the transcription activity of tumor suppressor protein p53. (Q34968621) (← links)
- Rapid Tumor Formation of Human T-Cell Leukemia Virus Type 1-Infected Cell Lines in Novel NOD-SCID/γcnullMice: Suppression by an Inhibitor against NF-κB (Q34975395) (← links)
- The human T-cell leukemia virus type 1 oncoprotein tax controls forkhead box O4 activity through degradation by the proteasome (Q35077555) (← links)
- Human T-cell lymphotropic virus: a model of NF-κB-associated tumorigenesis (Q35090534) (← links)
- Molecular determinants of melanoma malignancy: selecting targets for improved efficacy of chemotherapy (Q35118157) (← links)
- T-cell control by human T-cell leukemia/lymphoma virus type 1. (Q35611762) (← links)
- Activation of NF-kappaB by HTLV-I and implications for cell transformation (Q36254865) (← links)
- Impact of HTLV-I Tax on cell cycle progression and the cellular DNA damage repair response (Q36254875) (← links)
- PI3K/AKT inhibition induces caspase-dependent apoptosis in HTLV-1-transformed cells (Q36358632) (← links)
- Inhibition of methyltransferases results in induction of g2/m checkpoint and programmed cell death in human T-lymphotropic virus type 1-transformed cells (Q36424037) (← links)
- Wip1 and p53 contribute to HTLV-1 Tax-induced tumorigenesis (Q36494000) (← links)
- Human T-cell leukaemia virus type 1 (HTLV-1) infectivity and cellular transformation (Q36769835) (← links)
- Human T-cell leukemia virus type 1 infection leads to arrest in the G1 phase of the cell cycle (Q36845774) (← links)
- Small-molecule inhibitor which reactivates p53 in human T-cell leukemia virus type 1-transformed cells (Q36845802) (← links)
- Physical and in silico approaches identify DNA-PK in a Tax DNA-damage response interactome (Q36957291) (← links)
- HTLV-1 Tax oncoprotein subverts the cellular DNA damage response via binding to DNA-dependent protein kinase (Q37021560) (← links)
- Double-edged swords as cancer therapeutics: simultaneously targeting p53 and NF-kappaB pathways. (Q37337951) (← links)
- Apoptosis induced by the histone deacetylase inhibitor FR901228 in human T-cell leukemia virus type 1-infected T-cell lines and primary adult T-cell leukemia cells (Q37422407) (← links)
- Ubiquitination of human T-cell leukemia virus type 1 tax modulates its activity. (Q37583494) (← links)
- Targeting HTLV-1 activation of NFκB in mouse models and ATLL patients (Q37945228) (← links)
- HTLV-1-associated infective dermatitis: updates on the pathogenesis (Q38060839) (← links)
- Human T-lymphotropic virus proteins and post-translational modification pathways (Q38156859) (← links)
- Regulation of HTLV-1 tax stability, cellular trafficking and NF-κB activation by the ubiquitin-proteasome pathway (Q38262353) (← links)
- Human T-cell leukemia virus type 1 Tax attenuates gamma-irradiation-induced apoptosis through physical interaction with Chk2. (Q38321163) (← links)
- Id-1 expression promotes cell survival through activation of NF-kappaB signalling pathway in prostate cancer cells (Q38352246) (← links)
- Repression of IRF-4 target genes in human T cell leukemia virus-1 infection (Q38362324) (← links)