Pages that link to "Q37774243"

The following pages link to Glomerular diseases: genetic causes and future therapeutics. (Q37774243):

Displaying 29 items.

• Podocytes (Q26766118) (← links)
• MYO1E mutations and childhood familial focal segmental glomerulosclerosis (Q30541167) (← links)
• Xenopus as a model system for the study of GOLPH2/GP73 function: Xenopus GOLPH2 is required for pronephros development. (Q31068632) (← links)
• Using zebrafish to study podocyte genesis during kidney development and regeneration (Q34206971) (← links)
• MAGI-2 scaffold protein is critical for kidney barrier function (Q34384101) (← links)
• Next-generation sequencing to dissect hereditary nephrotic syndrome in mice identifies a hypomorphic mutation in Lamb2 and models Pierson's syndrome (Q34559353) (← links)
• Molecular Mechanisms of Podocyte Development Revealed by Zebrafish Kidney Research (Q34626179) (← links)
• The etiology of glomerulonephritis: roles of infection and autoimmunity (Q35118856) (← links)
• Alpha-actinin-4 and CLP36 protein deficiencies contribute to podocyte defects in multiple human glomerulopathies (Q35182978) (← links)
• Sirtuin1 Maintains Actin Cytoskeleton by Deacetylation of Cortactin in Injured Podocytes (Q35903079) (← links)
• Angiotensin II induces endoplasmic reticulum stress in podocyte, which would be further augmented by PI3-kinase inhibition (Q36572270) (← links)
• Genetic Ablation of Calcium-independent Phospholipase A2γ Induces Glomerular Injury in Mice. (Q37078637) (← links)
• Functional screening in Drosophila identifies Alzheimer's disease susceptibility genes and implicates Tau-mediated mechanisms (Q37508556) (← links)
• Childhood nephrotic syndrome--current and future therapies (Q38017937) (← links)
• Facing glycosphingolipid-Shiga toxin interaction: dire straits for endothelial cells of the human vasculature (Q38024274) (← links)
• The renal biopsy in the genomic era. (Q38062343) (← links)
• Proteostasis in endoplasmic reticulum--new mechanisms in kidney disease (Q38206090) (← links)
• Thrombin-Induced Podocyte Injury Is Protease-Activated Receptor Dependent (Q38829694) (← links)
• Stress Signal Network between Hypoxia and ER Stress in Chronic Kidney Disease (Q39147185) (← links)
• Deletion of inositol-requiring enzyme-1α in podocytes disrupts glomerular capillary integrity and autophagy (Q41813833) (← links)
• Angiopoietin-like 3 induces podocyte F-actin rearrangement through integrin α(V)β₃/FAK/PI3K pathway-mediated Rac1 activation (Q41865483) (← links)
• Podocyte Injury Associated with Mutant α-Actinin-4 (Q42844049) (← links)
• RNA-seq of serial kidney biopsies obtained during progression of chronic kidney disease from dogs with X-linked hereditary nephropathy. (Q47094474) (← links)
• Ste20-like Kinase, SLK, a Novel Mediator of Podocyte Integrity. (Q47571484) (← links)
• Endoplasmic reticulum stress, the unfolded protein response and autophagy in kidney diseases (Q47711878) (← links)
• Successful production of genome-edited rats by the rGONAD method. (Q52431211) (← links)
• Endoplasmic reticulum stress signal impairs erythropoietin production: a role for ATF4. (Q53129238) (← links)
• Interaction of receptor for advanced glycation end products with advanced oxidation protein products induces podocyte injury (Q84970924) (← links)
• The genetics of atypical hemolytic uremic syndrome (Q92730532) (← links)