Pages that link to "Q34346028"

The following pages link to Truncation of herpes simplex virus type 2 glycoprotein B increases its cell surface expression and activity in cell-cell fusion, but these properties are unrelated (Q34346028):

Displayed 41 items.

• Differences in the N termini of herpes simplex virus type 1 and 2 gDs that influence functional interactions with the human entry receptor Nectin-2 and an entry receptor expressed in Chinese hamster ovary cells (Q24315117) ‎ (← links)
• Herpes simplex virus type 1 mediates fusion through a hemifusion intermediate by sequential activity of glycoproteins D, H, L, and B (Q24682681) ‎ (← links)
• Random linker-insertion mutagenesis to identify functional domains of herpes simplex virus type 1 glycoprotein B (Q27480974) ‎ (← links)
• Characterization of Vesicular Stomatitis Virus Pseudotypes Bearing Essential Entry Glycoproteins gB, gD, gH, and gL of Herpes Simplex Virus 1. (Q30353044) ‎ (← links)
• Syncytial phenotype of C-terminally truncated herpes simplex virus type 1 gB is associated with diminished membrane interactions (Q33826866) ‎ (← links)
• Identification of functional domains in herpes simplex virus 2 glycoprotein B. (Q34546061) ‎ (← links)
• Modulation of Epstein-Barr virus glycoprotein B (gB) fusion activity by the gB cytoplasmic tail domain (Q34557882) ‎ (← links)
• Differential effects on cell fusion activity of mutations in herpes simplex virus 1 glycoprotein B (gB) dependent on whether a gD receptor or a gB receptor is overexpressed (Q34982160) ‎ (← links)
• Activation of Fusion by the SER Virus F Protein: a Low-pH-Dependent Paramyxovirus Entry Process (Q34997435) ‎ (← links)
• Role of the herpes simplex virus 1 Us3 kinase phosphorylation site and endocytosis motifs in the intracellular transport and neurovirulence of envelope glycoprotein B. (Q35076725) ‎ (← links)
• HSV-2- and HIV-1- permissive cell lines co-infected by HSV-2 and HIV-1 co-replicate HSV-2 and HIV-1 without production of HSV-2/HIV-1 pseudotype particles. (Q35585672) ‎ (← links)
• Herpesvirus interference with virus-specific antibodies: bridging antibodies, internalizing antibodies, and hiding from antibodies (Q36330380) ‎ (← links)
• The why's of Y-based motifs in alphaherpesvirus envelope proteins (Q36370795) ‎ (← links)
• An immunoreceptor tyrosine-based inhibition motif in varicella-zoster virus glycoprotein B regulates cell fusion and skin pathogenesis (Q36583285) ‎ (← links)
• Conservation of the glycoprotein B homologs of the Kaposi׳s sarcoma-associated herpesvirus (KSHV/HHV8) and old world primate rhadinoviruses of chimpanzees and macaques (Q36945437) ‎ (← links)
• Mutations in the cytoplasmic domain of the Newcastle disease virus fusion protein confer hyperfusogenic phenotypes modulating viral replication and pathogenicity (Q37123217) ‎ (← links)
• Cell-surface expression of a mutated Epstein-Barr virus glycoprotein B allows fusion independent of other viral proteins (Q37713781) ‎ (← links)
• Mutations in Pseudorabies Virus Glycoproteins gB, gD, and gH Functionally Compensate for the Absence of gL. (Q38812527) ‎ (← links)
• Contribution of N-linked glycans on HSV-2 gB to cell-cell fusion and viral entry (Q38876017) ‎ (← links)
• Residues within the C-terminal arm of the herpes simplex virus 1 glycoprotein B ectodomain contribute to its refolding during the fusion step of virus entry (Q39366614) ‎ (← links)
• Characterization of cytoplasmic motifs important in rhesus rhadinovirus gB processing and trafficking (Q39754233) ‎ (← links)
• Contribution of endocytic motifs in the cytoplasmic tail of herpes simplex virus type 1 glycoprotein B to virus replication and cell-cell fusion (Q40072572) ‎ (← links)
• Intracellular trafficking and maturation of herpes simplex virus type 1 gB and virus egress require functional biogenesis of multivesicular bodies (Q40096537) ‎ (← links)
• Alanine substitution of conserved residues in the cytoplasmic tail of herpes simplex virus gB can enhance or abolish cell fusion activity and viral entry (Q40344177) ‎ (← links)
• Pseudorabies virus glycoprotein gD contains a functional endocytosis motif that acts in concert with an endocytosis motif in gB to drive internalization of antibody-antigen complexes from the surface of infected monocytes (Q40423177) ‎ (← links)
• Effects of mutations in the cytoplasmic domain of herpes simplex virus type 1 glycoprotein B on intracellular transport and infectivity (Q40468692) ‎ (← links)
• Conserved cytoplasmic domain sequences mediate the ER export of VZV, HSV-1, and HCMV gB. (Q40513452) ‎ (← links)
• Internalization of pseudorabies virus glycoprotein B is mediated by an interaction between the YQRL motif in its cytoplasmic domain and the clathrin-associated AP-2 adaptor complex (Q40530724) ‎ (← links)
• Coexpression of UL20p and gK inhibits cell-cell fusion mediated by herpes simplex virus glycoproteins gD, gH-gL, and wild-type gB or an endocytosis-defective gB mutant and downmodulates their cell surface expression (Q40535818) ‎ (← links)
• Regulation of varicella-zoster virus-induced cell-to-cell fusion by the endocytosis-competent glycoproteins gH and gE. (Q40624826) ‎ (← links)
• Interplay between the Herpes Simplex Virus 1 gB Cytodomain and the gH Cytotail during Cell-Cell Fusion (Q40984579) ‎ (← links)
• Herpes simplex virus type 1 capsids transit by the trans-Golgi network, where viral glycoproteins accumulate independently of capsid egress. (Q42011253) ‎ (← links)
• Membrane requirement for folding of the herpes simplex virus 1 gB cytodomain suggests a unique mechanism of fusion regulation (Q42280908) ‎ (← links)
• Multifaceted sequence-dependent and -independent roles for reovirus FAST protein cytoplasmic tails in fusion pore formation and syncytiogenesis. (Q42689581) ‎ (← links)
• Optimal replication of human cytomegalovirus correlates with endocytosis of glycoprotein gpUL132. (Q42737597) ‎ (← links)
• Identification of syncytial mutations in a clinical isolate of herpes simplex virus 2. (Q45569727) ‎ (← links)
• Fusion activity of lipid-anchored envelope glycoproteins of herpes simplex virus type 1. (Q45630446) ‎ (← links)
• Functional relevance of the transmembrane domain and cytoplasmic tail of the pseudorabies virus glycoprotein H for membrane fusion. (Q52331818) ‎ (← links)
• Natural Selection of Glycoprotein B Mutations That Rescue the Small-Plaque Phenotype of a Fusion-Impaired Herpes Simplex Virus Mutant (Q57459069) ‎ (← links)
• Structural basis for membrane anchoring and fusion regulation of the herpes simplex virus fusogen gB (Q59348998) ‎ (← links)
• The structural basis of herpesvirus entry (Q100762135) ‎ (← links)