Pages that link to "Q34346028"
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The following pages link to Truncation of herpes simplex virus type 2 glycoprotein B increases its cell surface expression and activity in cell-cell fusion, but these properties are unrelated (Q34346028):
Displayed 41 items.
- Differences in the N termini of herpes simplex virus type 1 and 2 gDs that influence functional interactions with the human entry receptor Nectin-2 and an entry receptor expressed in Chinese hamster ovary cells (Q24315117) (← links)
- Herpes simplex virus type 1 mediates fusion through a hemifusion intermediate by sequential activity of glycoproteins D, H, L, and B (Q24682681) (← links)
- Random linker-insertion mutagenesis to identify functional domains of herpes simplex virus type 1 glycoprotein B (Q27480974) (← links)
- Characterization of Vesicular Stomatitis Virus Pseudotypes Bearing Essential Entry Glycoproteins gB, gD, gH, and gL of Herpes Simplex Virus 1. (Q30353044) (← links)
- Syncytial phenotype of C-terminally truncated herpes simplex virus type 1 gB is associated with diminished membrane interactions (Q33826866) (← links)
- Identification of functional domains in herpes simplex virus 2 glycoprotein B. (Q34546061) (← links)
- Modulation of Epstein-Barr virus glycoprotein B (gB) fusion activity by the gB cytoplasmic tail domain (Q34557882) (← links)
- Differential effects on cell fusion activity of mutations in herpes simplex virus 1 glycoprotein B (gB) dependent on whether a gD receptor or a gB receptor is overexpressed (Q34982160) (← links)
- Activation of Fusion by the SER Virus F Protein: a Low-pH-Dependent Paramyxovirus Entry Process (Q34997435) (← links)
- Role of the herpes simplex virus 1 Us3 kinase phosphorylation site and endocytosis motifs in the intracellular transport and neurovirulence of envelope glycoprotein B. (Q35076725) (← links)
- HSV-2- and HIV-1- permissive cell lines co-infected by HSV-2 and HIV-1 co-replicate HSV-2 and HIV-1 without production of HSV-2/HIV-1 pseudotype particles. (Q35585672) (← links)
- Herpesvirus interference with virus-specific antibodies: bridging antibodies, internalizing antibodies, and hiding from antibodies (Q36330380) (← links)
- The why's of Y-based motifs in alphaherpesvirus envelope proteins (Q36370795) (← links)
- An immunoreceptor tyrosine-based inhibition motif in varicella-zoster virus glycoprotein B regulates cell fusion and skin pathogenesis (Q36583285) (← links)
- Conservation of the glycoprotein B homologs of the Kaposi׳s sarcoma-associated herpesvirus (KSHV/HHV8) and old world primate rhadinoviruses of chimpanzees and macaques (Q36945437) (← links)
- Mutations in the cytoplasmic domain of the Newcastle disease virus fusion protein confer hyperfusogenic phenotypes modulating viral replication and pathogenicity (Q37123217) (← links)
- Cell-surface expression of a mutated Epstein-Barr virus glycoprotein B allows fusion independent of other viral proteins (Q37713781) (← links)
- Mutations in Pseudorabies Virus Glycoproteins gB, gD, and gH Functionally Compensate for the Absence of gL. (Q38812527) (← links)
- Contribution of N-linked glycans on HSV-2 gB to cell-cell fusion and viral entry (Q38876017) (← links)
- Residues within the C-terminal arm of the herpes simplex virus 1 glycoprotein B ectodomain contribute to its refolding during the fusion step of virus entry (Q39366614) (← links)
- Characterization of cytoplasmic motifs important in rhesus rhadinovirus gB processing and trafficking (Q39754233) (← links)
- Contribution of endocytic motifs in the cytoplasmic tail of herpes simplex virus type 1 glycoprotein B to virus replication and cell-cell fusion (Q40072572) (← links)
- Intracellular trafficking and maturation of herpes simplex virus type 1 gB and virus egress require functional biogenesis of multivesicular bodies (Q40096537) (← links)
- Alanine substitution of conserved residues in the cytoplasmic tail of herpes simplex virus gB can enhance or abolish cell fusion activity and viral entry (Q40344177) (← links)
- Pseudorabies virus glycoprotein gD contains a functional endocytosis motif that acts in concert with an endocytosis motif in gB to drive internalization of antibody-antigen complexes from the surface of infected monocytes (Q40423177) (← links)
- Effects of mutations in the cytoplasmic domain of herpes simplex virus type 1 glycoprotein B on intracellular transport and infectivity (Q40468692) (← links)
- Conserved cytoplasmic domain sequences mediate the ER export of VZV, HSV-1, and HCMV gB. (Q40513452) (← links)
- Internalization of pseudorabies virus glycoprotein B is mediated by an interaction between the YQRL motif in its cytoplasmic domain and the clathrin-associated AP-2 adaptor complex (Q40530724) (← links)
- Coexpression of UL20p and gK inhibits cell-cell fusion mediated by herpes simplex virus glycoproteins gD, gH-gL, and wild-type gB or an endocytosis-defective gB mutant and downmodulates their cell surface expression (Q40535818) (← links)
- Regulation of varicella-zoster virus-induced cell-to-cell fusion by the endocytosis-competent glycoproteins gH and gE. (Q40624826) (← links)
- Interplay between the Herpes Simplex Virus 1 gB Cytodomain and the gH Cytotail during Cell-Cell Fusion (Q40984579) (← links)
- Herpes simplex virus type 1 capsids transit by the trans-Golgi network, where viral glycoproteins accumulate independently of capsid egress. (Q42011253) (← links)
- Membrane requirement for folding of the herpes simplex virus 1 gB cytodomain suggests a unique mechanism of fusion regulation (Q42280908) (← links)
- Multifaceted sequence-dependent and -independent roles for reovirus FAST protein cytoplasmic tails in fusion pore formation and syncytiogenesis. (Q42689581) (← links)
- Optimal replication of human cytomegalovirus correlates with endocytosis of glycoprotein gpUL132. (Q42737597) (← links)
- Identification of syncytial mutations in a clinical isolate of herpes simplex virus 2. (Q45569727) (← links)
- Fusion activity of lipid-anchored envelope glycoproteins of herpes simplex virus type 1. (Q45630446) (← links)
- Functional relevance of the transmembrane domain and cytoplasmic tail of the pseudorabies virus glycoprotein H for membrane fusion. (Q52331818) (← links)
- Natural Selection of Glycoprotein B Mutations That Rescue the Small-Plaque Phenotype of a Fusion-Impaired Herpes Simplex Virus Mutant (Q57459069) (← links)
- Structural basis for membrane anchoring and fusion regulation of the herpes simplex virus fusogen gB (Q59348998) (← links)
- The structural basis of herpesvirus entry (Q100762135) (← links)