Pages that link to "Q28589793"
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The following pages link to Changes in cortical and striatal neurons predict behavioral and electrophysiological abnormalities in a transgenic murine model of Huntington's disease (Q28589793):
Displaying 50 items.
- BCL2-associated X protein (Q14905404) (← links)
- Interaction of the nuclear matrix protein NAKAP with HypA and huntingtin: implications for nuclear toxicity in Huntington's disease pathogenesis (Q24300257) (← links)
- Age-dependent alterations of corticostriatal activity in the YAC128 mouse model of Huntington disease (Q24657764) (← links)
- Role of cerebral cortex in the neuropathology of Huntington's disease (Q26829211) (← links)
- Mouse models of polyglutamine diseases: review and data table. Part I (Q26853025) (← links)
- Pluripotent stem cells models for Huntington's disease: prospects and challenges (Q26860151) (← links)
- Disrupted motor learning and long-term synaptic plasticity in mice lacking NMDAR1 in the striatum (Q28592654) (← links)
- Onset and progression of behavioral and molecular phenotypes in a novel congenic R6/2 line exhibiting intergenerational CAG repeat stability (Q28741746) (← links)
- Induction of neostriatal neurogenesis slows disease progression in a transgenic murine model of Huntington disease (Q30480065) (← links)
- Mouse models of Huntington's disease and methodological considerations for therapeutic trials (Q30481208) (← links)
- Neurexophilin 3 is highly localized in cortical and cerebellar regions and is functionally important for sensorimotor gating and motor coordination (Q30499012) (← links)
- Functional imaging in Huntington's disease (Q33403110) (← links)
- Progressive synaptic pathology of motor cortical neurons in a BAC transgenic mouse model of Huntington's disease (Q33573371) (← links)
- Complexity and heterogeneity: what drives the ever-changing brain in Huntington's disease? (Q33619310) (← links)
- Bax deletion prevents neuronal loss but not neurological symptoms in a transgenic model of inherited prion disease. (Q33715082) (← links)
- Transcriptional changes in Huntington disease identified using genome-wide expression profiling and cross-platform analysis (Q33755822) (← links)
- Huntingtin localisation studies - a technical review (Q33826159) (← links)
- Differential vulnerability of neurons in Huntington's disease: the role of cell type-specific features (Q33931283) (← links)
- The energetics of Huntington's disease (Q34308028) (← links)
- Lessons from animal models of Huntington's disease (Q34586943) (← links)
- The role of chaperones in polyglutamine disease (Q34688320) (← links)
- Huntingtin-interacting protein 1 influences worm and mouse presynaptic function and protects Caenorhabditis elegans neurons against mutant polyglutamine toxicity. (Q34700142) (← links)
- Mouse and fly models of neurodegeneration (Q34783822) (← links)
- Transgenic animal models of neurodegeneration based on human genetic studies. (Q34890228) (← links)
- Huntington's disease: the mystery unfolds? (Q35038660) (← links)
- Functional Changes in Neocortical Activity in Huntington's Disease Model Mice: An in vivo Intracellular Study (Q35055109) (← links)
- Impaired alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor trafficking and function by mutant huntingtin. (Q35311070) (← links)
- Conditional BDNF release under pathological conditions improves Huntington's disease pathology by delaying neuronal dysfunction (Q35487248) (← links)
- Altered excitatory and inhibitory inputs to striatal medium-sized spiny neurons and cortical pyramidal neurons in the Q175 mouse model of Huntington's disease (Q35560350) (← links)
- An anticholinergic reverses motor control and corticostriatal LTD deficits in Dyt1 ΔGAG knock-in mice (Q35576523) (← links)
- Neurotrophic factors in Huntington's disease (Q35618345) (← links)
- Genetic and environmental factors in the pathogenesis of Huntington's disease (Q35639868) (← links)
- Mutant huntingtin, abnormal mitochondrial dynamics, defective axonal transport of mitochondria, and selective synaptic degeneration in Huntington's disease (Q35645271) (← links)
- CAG repeat lengths > or =335 attenuate the phenotype in the R6/2 Huntington's disease transgenic mouse (Q35703870) (← links)
- Striatal atrophy and dendritic alterations in a knock-in mouse model of Huntington's disease (Q35871006) (← links)
- Glutamate receptor abnormalities in the YAC128 transgenic mouse model of Huntington's disease (Q36016647) (← links)
- Therapeutic silencing of mutant huntingtin with siRNA attenuates striatal and cortical neuropathology and behavioral deficits (Q36089160) (← links)
- A role for Kalirin-7 in corticostriatal synaptic dysfunction in Huntington's disease (Q36331582) (← links)
- Suppression of neurodegeneration and increased neurotransmission caused by expanded full-length huntingtin accumulating in the cytoplasm (Q36514656) (← links)
- Selective neuronal degeneration in Huntington's disease (Q36597920) (← links)
- Elevated NADPH oxidase activity contributes to oxidative stress and cell death in Huntington's disease (Q36626877) (← links)
- The corticostriatal pathway in Huntington's disease (Q36684263) (← links)
- Multiple pathways contribute to the pathogenesis of Huntington disease (Q36686156) (← links)
- Up-regulation of GLT1 expression increases glutamate uptake and attenuates the Huntington's disease phenotype in the R6/2 mouse (Q36713909) (← links)
- Characterization of forebrain neurons derived from late-onset Huntington's disease human embryonic stem cell lines. (Q36742638) (← links)
- Regulation of feedback between protein kinase A and the proteasome system worsens Huntington's disease (Q36757044) (← links)
- Drug targeting of dysregulated transcription in Huntington's disease (Q36767264) (← links)
- Full length mutant huntingtin is required for altered Ca2+ signaling and apoptosis of striatal neurons in the YAC mouse model of Huntington's disease. (Q36861129) (← links)
- Multiple sources of striatal inhibition are differentially affected in Huntington's disease mouse models (Q36940345) (← links)
- Dysregulated information processing by medium spiny neurons in striatum of freely behaving mouse models of Huntington's disease (Q36956907) (← links)