Pages that link to "Q28299813"
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The following pages link to Protective effects of antiarrhythmic agents against anoxic injury in CNS white matter (Q28299813):
Displaying 13 items.
- Sodium MRI of multiple sclerosis (Q30926519) (← links)
- Anoxic and ischemic injury of myelinated axons in CNS white matter: from mechanistic concepts to therapeutics (Q34452140) (← links)
- Optic nerve and neuroprotection strategies (Q35944015) (← links)
- Mechanisms of disease: sodium channels and neuroprotection in multiple sclerosis-current status (Q37070221) (← links)
- Experimental neuronal protection in cerebral ischaemia Part II: Potential neuroprotective drugs (Q37220928) (← links)
- Na(+)-Ca2+ exchange in anoxic/ischemic injury of CNS myelinated axons (Q41003748) (← links)
- Modulation of angiogenesis by dithiolethione-modified NSAIDs and valproic acid. (Q41556578) (← links)
- The use-dependent sodium channel blocker mexiletine is neuroprotective against global ischemic injury (Q43577555) (← links)
- Differential effects of bupivacaine enantiomers, ropivacaine and lidocaine on up-regulation of cell surface voltage-dependent sodium channels in adrenal chromaffin cells (Q44346558) (← links)
- Clinical applications of ultra-high field magnetic resonance imaging in multiple sclerosis. (Q47840104) (← links)
- Sodium channel modulators prevent oxygen and glucose deprivation injury and glutamate release in rat neocortical cultures (Q48643826) (← links)
- Local blockade of sodium channels by tetrodotoxin ameliorates tissue loss and long-term functional deficits resulting from experimental spinal cord injury. (Q51542251) (← links)
- Mechanisms of ischaemic damage to central white matter axons: a quantitative histological analysis using rat optic nerve (Q73333079) (← links)