Pages that link to "Q27490541"
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The following pages link to Effect of Killer Immunoglobulin-Like Receptors in the Response to Combined Treatment in Patients with Chronic Hepatitis C Virus Infection (Q27490541):
Displayed 41 items.
- Natural killer cell responses during viral infections: flexibility and conditioning of innate immunity by experience (Q26850968) (← links)
- HTLV-1/-2 and HIV-1 co-infections: retroviral interference on host immune status (Q26866444) (← links)
- IL28B, HLA-C, and KIR variants additively predict response to therapy in chronic hepatitis C virus infection in a European Cohort: a cross-sectional study (Q28477057) (← links)
- KIR-HLA genotypes in HIV-infected patients lacking immunological recovery despite effective antiretroviral therapy (Q28477797) (← links)
- Natural killer KIR3DS1 is closely associated with HCV viral clearance and sustained virological response in HIV/HCV patients (Q31115968) (← links)
- Long-term effect on natural killer cells by interferon-α therapy on the outcomes of HCV infection (Q33588171) (← links)
- Natural killer cell-based therapies (Q34988727) (← links)
- KIR/HLA interactions and pathogen immunity (Q35001169) (← links)
- NK cells, innate immunity and hepatitis C infection after liver transplantation (Q35019869) (← links)
- KIR, HLA, and IL28B variant predict response to antiviral therapy in genotype 1 chronic hepatitis C patients in Japan (Q35070933) (← links)
- CD4saurus Rex &HIVelociraptor vs. development of clinically useful immunological markers: a Jurassic tale of frozen evolution (Q35121645) (← links)
- Killer-cell immunoglobulin-like receptor genotyping and HLA killer-cell immunoglobulin-like receptor-ligand identification by real-time polymerase chain reaction (Q35148027) (← links)
- Inherent transcriptional signatures of NK cells are associated with response to IFNα + rivabirin therapy in patients with Hepatitis C Virus (Q35158581) (← links)
- Diversity of killer cell immunoglobulin-like receptor (KIR) genotypes and KIR2DL2/3 variants in HCV treatment outcome (Q35187793) (← links)
- A polymorphism in IL28B distinguishes exposed, uninfected individuals from spontaneous resolvers of HCV infection. (Q35356926) (← links)
- Frequency of killer cell immunoglobulin-like receptors (KIRs) in Korean patients with chronic HCV infection (Q35511598) (← links)
- NK cells: immune cross-talk and therapeutic implications (Q35591444) (← links)
- Genetically associated CD16(+)56(-) natural killer cell interferon (IFN)-αR expression regulates signaling and is implicated in IFN-α-induced hepatitis C virus decline (Q35808616) (← links)
- Glances in Immunology of HIV and HCV Infection (Q36032051) (← links)
- IL-28B Genetic Variants Determine the Extent of Monocyte-Induced Activation of NK Cells in Hepatitis C. (Q37226696) (← links)
- Individualization of chronic hepatitis C treatment according to the host characteristics (Q37654502) (← links)
- Natural cytotoxicity receptor-dependent natural killer cytolytic activity directed at hepatitis C Virus (HCV) is associated with liver inflammation, African American race, IL28B genotype, and response to pegylated interferon/ribavirin therapy in chr (Q37719750) (← links)
- Host genetics in immune-mediated hepatitis C virus clearance (Q37862746) (← links)
- Maternal KIR and fetal HLA-C: a fine balance (Q37923237) (← links)
- Hepatitis C virus-mediated modulation of cellular immunity (Q38036576) (← links)
- Natural killer cells in hepatitis C virus infection. (Q38061192) (← links)
- Innate and adaptive genetic pathways in HCV infection (Q38364524) (← links)
- NK cells: tuned by peptide? (Q38568884) (← links)
- Association of killer cell immunoglobulin-like receptor polymorphisms with chronic hepatitis C and responses to therapy in Brazil (Q39555726) (← links)
- Peptide selectivity discriminates NK cells from KIR2DL2- and KIR2DL3-positive individuals (Q41682815) (← links)
- Comparative analysis of NK-cell receptor expression and function across primate species: Perspective on antiviral defenses (Q41993469) (← links)
- Exploration of genetically determined resistance against hepatitis C infection in high-risk injecting drug users. (Q42232714) (← links)
- Activating NK cell receptor expression/function (NKp30, NKp46, DNAM-1) during chronic viraemic HCV infection is associated with the outcome of combined treatment (Q43037932) (← links)
- Killer cell immunoglobulin-like receptors and response to antiviral treatment in Thai patients with chronic hepatitis C virus genotype 3a. (Q43038593) (← links)
- Killer cell immunoglobulin-like receptor (KIR) gene diversity in a population naturally exposed to malaria in Porto Velho, Northern Brazil. (Q44397350) (← links)
- IL-12 and IL-15 induce the expression of CXCR6 and CD49a on peripheral natural killer cells. (Q47738030) (← links)
- Higher expression of inhibitory CD158b and CD158e NK cell receptor and age predicts treatment response in children with chronic hepatitis C. (Q48337795) (← links)
- KIR2DL2 combined with HLA-C1 confers risk of hepatitis C virus-related hepatocellular carcinoma in younger patients. (Q55311659) (← links)
- Evolving Trends in the Hepatitis C Virus Molecular Epidemiology Studies: From the Viral Sequences to the Human Genome (Q58701375) (← links)
- KIR2DL2/C1 is a Risk Factor for Chronic Infection and Associated with Non-response to PEG-IFN and RBV Combination Therapy in Hepatitis C Virus Genotype 1b Patients in China (Q59356136) (← links)
- Presence of donor-encoded centromeric KIR B content increases the risk of infectious mortality in recipients of myeloablative, T-cell deplete, HLA-matched HCT to treat AML (Q90590311) (← links)