Pages that link to "Q24313261"
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The following pages link to Mechanisms of STAT protein activation by oncogenic KIT mutants in neoplastic mast cells (Q24313261):
Displaying 29 items.
- KIT proto-oncogene, receptor tyrosine kinase (Q909409) (← links)
- Allosteric pathway identification through network analysis: from molecular dynamics simulations to interactive 2D and 3D graphs. (Q30368055) (← links)
- Towards a quantitative understanding of the MITF-PIAS3-STAT3 connection (Q34153534) (← links)
- Oncogenic Kit signals on endolysosomes and endoplasmic reticulum are essential for neoplastic mast cell proliferation (Q34833416) (← links)
- Novel mechanism for Fc{epsilon}RI-mediated signal transducer and activator of transcription 5 (STAT5) tyrosine phosphorylation and the selective influence of STAT5B over mast cell cytokine production (Q35694030) (← links)
- Mastocytosis: a mutated KIT receptor induced myeloproliferative disorder (Q36210845) (← links)
- M-COPA suppresses endolysosomal Kit-Akt oncogenic signalling through inhibiting the secretory pathway in neoplastic mast cells. (Q36344903) (← links)
- TAK1 and IKK2, novel mediators of SCF-induced signaling and potential targets for c-Kit-driven diseases. (Q36557145) (← links)
- Update on diagnosis and treatment of mastocytosis (Q37869373) (← links)
- STAT5 acetylation: Mechanisms and consequences for immunological control and leukemogenesis (Q38177821) (← links)
- Co-operating STAT5 and AKT signaling pathways in chronic myeloid leukemia and mastocytosis: possible new targets of therapy (Q38193659) (← links)
- The activation loop tyrosine 823 is essential for the transforming capacity of the c-Kit oncogenic mutant D816V. (Q38936739) (← links)
- Distinct signalling pathways for mutated KIT(V560G) and KIT(D816V) in mastocytosis (Q39137990) (← links)
- KIT-D816V oncogenic activity is controlled by the juxtamembrane docking site Y568-Y570. (Q39193013) (← links)
- An essential pathway links FLT3-ITD, HCK and CDK6 in acute myeloid leukemia (Q41819724) (← links)
- Alterations of oncogenes expression in NK cells in patients with cancer (Q44717067) (← links)
- Drug-induced inhibition of phosphorylation of STAT5 overrides drug resistance in neoplastic mast cells. (Q48104415) (← links)
- Direct engagement of the PI3K pathway by mutant KIT dominates oncogenic signaling in gastrointestinal stromal tumor. (Q48327749) (← links)
- The role of regulatory T cells and genes involved in their differentiation in pathogenesis of selected inflammatory and neoplastic skin diseases. Part III: Polymorphisms of genes involved in Tregs' activation and function. (Q49391905) (← links)
- Overexpression of P-glycoprotein, STAT3, phospho-STAT3 and KIT in spontaneous canine cutaneous mast cell tumours before and after prednisolone treatment. (Q54524879) (← links)
- Annotating activation/inhibition relationships to protein-protein interactions using gene ontology relations. (Q55257940) (← links)
- A pan-cancer analysis of driver gene mutations, DNA methylation and gene expressions reveals that chromatin remodeling is a major mechanism inducing global changes in cancer epigenomes (Q58605511) (← links)
- Preclinical human models and emerging therapeutics for advanced systemic mastocytosis (Q59810100) (← links)
- Oncogenic KIT mutations induce STAT3-dependent autophagy to support cell proliferation in acute myeloid leukemia (Q91936391) (← links)
- Companion Animals as Models for Inhibition of STAT3 and STAT5 (Q92151739) (← links)
- Thymic Stromal Lymphopoietin Interferes with the Apoptosis of Human Skin Mast Cells by a Dual Strategy Involving STAT5/Mcl-1 and JNK/Bcl-xL (Q92454394) (← links)
- Pharmacological Inhibition of Oncogenic STAT3 and STAT5 Signaling in Hematopoietic Cancers (Q92814428) (← links)
- Virus-host interactome and proteomic survey of PBMCs from COVID-19 patients reveal potential virulence factors influencing SARS-CoV-2 pathogenesis (Q97940518) (← links)
- The V654A second-site KIT mutation increases tumor oncogenesis and STAT activation in a mouse model of gastrointestinal stromal tumor (Q100465046) (← links)