Aberrant RNA processing in a neurodegenerative disease: the cause for absent EAAT2, a glutamate transporter, in amyotrophic lateral sclerosis (Q28267385): Difference between revisions

@@ label / nl / label / nl @@
+Aberrant RNA processing in a neurodegenerative disease: the cause for absent EAAT2, a glutamate transporter, in amyotrophic lateral sclerosis
@@ label / ast / label / ast @@
+Aberrant RNA processing in a neurodegenerative disease: the cause for absent EAAT2, a glutamate transporter, in amyotrophic lateral sclerosis
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+wetenschappelijk artikel (gepubliceerd in 1998-03)
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+թվականի մարտին հրատարակված գիտական հոդված
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+სამეცნიერო სტატია
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@@ description / fi / description / fi @@
+tieteellinen artikkeli
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+מאמר מדעי
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+мақолаи илмӣ
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+научни чланак
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+թուականի Մարտին հրատարակուած գիտական յօդուած
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+سائنسی مضمون
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+مقالهٔ علمی
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@@ Property / main subject: amyotrophic lateral sclerosis / rank @@
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+Normal rank
@@ Property / main subject: neurodegeneration / reference @@
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@@ Property / cites work @@
+Mutations in the glutamate transporter EAAT2 gene do not cause abnormal EAAT2 transcripts in amyotrophic lateral sclerosis
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Evidence for autoimmunity in amyotrophic lateral sclerosis
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Functional comparisons of three glutamate transporter subtypes cloned from human motor cortex
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Increased 3-nitrotyrosine in both sporadic and familial amyotrophic lateral sclerosis
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+A controlled trial of riluzole in amyotrophic lateral sclerosis. ALS/Riluzole Study Group
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Superoxide dismutase activity, oxidative damage, and mitochondrial energy metabolism in familial and sporadic amyotrophic lateral sclerosis
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Superoxide dismutase concentration and activity in familial amyotrophic lateral sclerosis
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Glutamate transporter gene expression in amyotrophic lateral sclerosis motor cortex
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+El Escorial World Federation of Neurology criteria for the diagnosis of amyotrophic lateral sclerosis. Subcommittee on Motor Neuron Diseases/Amyotrophic Lateral Sclerosis of the World Federation of Neurology Research Group on Neuromuscular Diseases a
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Superoxide dismutase and familial amyotrophic lateral sclerosis: new insights into mechanisms and treatments
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Mechanisms of selective motor neuron death in ALS: insights from transgenic mouse models of motor neuron disease
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+NMDA and kainate induce internucleosomal DNA cleavage associated with both apoptotic and necrotic cell death in the neonatal rat brain
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+In vitro kainate injury to large, SMI-32(+) spinal neurons is Ca2+ dependent.
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Cell culture evidence for neuronal degeneration in amyotrophic lateral sclerosis being linked to glutamate AMPA/kainate receptors
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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 Before
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+DNA strand breaks induced by sustained glutamate excitotoxicity in primary neuronal cultures.
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Intron retention generates a novel isoform of the murine vitamin D receptor that acts in a dominant negative way on the vitamin D signaling pathway
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+An excitatory amino-acid transporter with properties of a ligand-gated chloride channel
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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 Before
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@@ Property / cites work @@
+Evidence of increased oxidative damage in both sporadic and familial amyotrophic lateral sclerosis
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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 Before
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@@ Property / cites work @@
+Increased 3-nitrotyrosine and oxidative damage in mice with a human copper/zinc superoxide dismutase mutation.
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+The SMN-SIP1 complex has an essential role in spliceosomal snRNP biogenesis
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Cellular and synaptic localization of the neuronal glutamate transporters excitatory amino acid transporter 3 and 4.
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+day
 Before
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@@ Property / cites work @@
+Benefit of vitamin E, riluzole, and gabapentin in a transgenic model of familial amyotrophic lateral sclerosis
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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@@ Property / cites work @@
+Genomic organization, promoter analysis, and chromosomal localization of the gene for the mouse glial high-affinity glutamate transporter Slc1a3.
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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++2021-01-07T00:00:00Z
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 Before
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@@ Property / cites work @@
+Brain glutamate transporter proteins form homomultimers
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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@@ Property / cites work @@
+Motor neuron degeneration induced by excitotoxin agonists has features in common with those seen in the SOD-1 transgenic mouse model of amyotrophic lateral sclerosis
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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@@ Property / cites work @@
+Parvalbumin and calbindin D-28k in the human motor system and in motor neuron disease
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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@@ Property / cites work @@
+An intronic mutation in a lariat branchpoint sequence is a direct cause of an inherited human disorder (fish-eye disease).
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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@@ Property / cites work @@
+Dose-ranging study of riluzole in amyotrophic lateral sclerosis. Amyotrophic Lateral Sclerosis/Riluzole Study Group II
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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@@ Property / cites work @@
+A mutant neurofilament subunit causes massive, selective motor neuron death: implications for the pathogenesis of human motor neuron disease
+Normal rank
+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Differential expression of two glial glutamate transporters in the rat brain: quantitative and immunocytochemical observations
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+The spinal muscular atrophy disease gene product, SMN, and its associated protein SIP1 are in a complex with spliceosomal snRNP proteins
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+Genomic organization of the human excitatory amino acid transporter gene GLT-1.
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+Placebo-controlled trial of gabapentin in patients with amyotrophic lateral sclerosis. WALS Study Group. Western Amyotrophic Lateral Sclerosis Study Group
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+smg mutants affect the expression of alternatively spliced SR protein mRNAs in Caenorhabditis elegans
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+Excitotoxicity hypothesis
@@ Property / cites work: Excitotoxicity hypothesis / rank @@
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@@ Property / cites work: Excitotoxicity hypothesis / reference @@
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Abnormal excitatory amino acid metabolism in amyotrophic lateral sclerosis
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Decreased glutamate transport by the brain and spinal cord in amyotrophic lateral sclerosis
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+Chronic inhibition of glutamate uptake produces a model of slow neurotoxicity
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+Localization of neuronal and glial glutamate transporters
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+Selective loss of glial glutamate transporter GLT‐1 in amyotrophic lateral sclerosis
+Normal rank
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+Knockout of glutamate transporters reveals a major role for astroglial transport in excitotoxicity and clearance of glutamate
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Excitotoxicity and motor neurone disease: a review of the evidence
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+[3H]d-aspartate binding sites in the normal human spinal cord and changes in motor neuron disease: a quantitative autoradiographic study
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+CSF and plasma amino acid levels in motor neuron disease: elevation of CSF glutamate in a subset of patients
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+Genetics of amyotrophic lateral sclerosis
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+stated in: Crossref
reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Serum antibodies to L-type calcium channels in patients with amyotrophic lateral sclerosis
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Human high affinity, Na(+)-dependent L-glutamate/L-aspartate transporter GLAST-1 (EAAT-1): gene structure and localization to chromosome 5p11-p12
+Normal rank
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+Epilepsy and exacerbation of brain injury in mice lacking the glutamate transporter GLT-1
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+Peroxynitrite inhibits glutamate transporter subtypes
+Normal rank
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+Alternative splicing in COL1A1 mRNA leads to a partial null allele and two In-frame forms with structural defects in non-lethal osteogenesis imperfecta
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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+An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria
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reference URL: https://api.crossref.org/works/10.1016%2FS0896-6273%2800%2980997-6
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Latest revision as of 20:34, 18 April 2022

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Language	Label	Description	Also known as
English	Aberrant RNA processing in a neurodegenerative disease: the cause for absent EAAT2, a glutamate transporter, in amyotrophic lateral sclerosis	scientific article

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Aberrant RNA processing in a neurodegenerative disease: the cause for absent EAAT2, a glutamate transporter, in amyotrophic lateral sclerosis (English)

Aberrant RNA processing in a neurodegenerative disease: the cause for absent EAAT2, a glutamate transporter, in amyotrophic lateral sclerosis (Q28267385): Difference between revisions

Latest revision as of 20:34, 18 April 2022

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