(Q46899191)

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Alternative macrophage activation is essential for survival during schistosomiasis and downmodulates T helper 1 responses and immunopathology.

scientific article published in May 2004

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28 December 2019

Alternative macrophage activation is essential for survival during schistosomiasis and downmodulates T helper 1 responses and immunopathology (English)

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28 December 2019

schistosomiasis

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Irmgard Förster

12

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28 December 2019

Magdalena Radwanska

6

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28 December 2019

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Horst Mossmann

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28 December 2019

Christoph Hölscher

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Christoph Hölscher

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28 December 2019

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De'Broski R Herbert

1

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28 December 2019

Markus Mohrs

3

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28 December 2019

Berenice Arendse

4

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28 December 2019

Anita Schwegmann

5

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28 December 2019

Mosiuoa Leeto

7

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28 December 2019

Richard Kirsch

8

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28 December 2019

Pauline Hall

9

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28 December 2019

Björn Claussen

11

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28 December 2019

Frank Brombacher

13

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28 December 2019

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1 May 2004

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28 December 2019

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28 December 2019

20

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28 December 2019

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28 December 2019

623-635

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28 December 2019

Differences between IL-4R alpha-deficient and IL-4-deficient mice reveal a role for IL-13 in the regulation of Th2 responses

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Somatostatin and intestinal schistosomiasis: therapeutic and neuropathological implications in host-parasite interactions

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An IL-13 inhibitor blocks the development of hepatic fibrosis during a T-helper type 2-dominated inflammatory response

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Conditional gene targeting in macrophages and granulocytes using LysMcre mice.

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Schistosome infection of transgenic mice defines distinct and contrasting pathogenic roles for IL-4 and IL-13: IL-13 is a profibrotic agent

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IL-4 plays a crucial role in regulating oxidative damage in the liver during schistosomiasis

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Alternative activation of macrophages

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IL-10 and the dangers of immune polarization: excessive type 1 and type 2 cytokine responses induce distinct forms of lethal immunopathology in murine schistosomiasis

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IL-4 receptor signaling is required for mannose receptor expression by macrophages recruited to granulomata but not resident cells in mice infected with Schistosoma mansoni

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IL-4 dependent alternatively-activated macrophages have a distinctive in vivo gene expression phenotype

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Interleukin-4 receptor alpha-deficient BALB/c mice show an unimpaired T helper 2 polarization in response to Leishmania major infection

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Central role for interleukin-4 in regulating nitric oxide-mediated inhibition of T-cell proliferation and gamma interferon production in schistosomiasis

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Identifiers

10.1016/S1074-7613(04)00107-4

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28 December 2019

PubMed publication ID

1 reference

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28 December 2019

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