(Q45422942)

14 December 2017

title

Decreasing the frameshift efficiency translates into an equivalent reduction of the replication of the human immunodeficiency virus type 1. (English)

1 reference

14 December 2017

4

Léa Brakier-Gingras

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Nikolaus Heveker

5

object named as

Nikolaus Heveker

1 reference

14 December 2017

Dominic Dulude

1

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14 December 2017

Yamina A Berchiche

2

1 reference

14 December 2017

Karine Gendron

3

1 reference

14 December 2017

Léa Brakier-Gingras

4

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14 December 2017

language of work or name

English

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publication date

25 October 2005

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14 December 2017

number of pages

10

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based on heuristic

inferred from page(s)

published in

Virology

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14 December 2017

volume

345

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14 December 2017

issue

1

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14 December 2017

page(s)

127-136

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In vitro characterization of a simian immunodeficiency virus-human immunodeficiency virus (HIV) chimera expressing HIV type 1 reverse transcriptase to study antiviral resistance in pigtail macaques

14 December 2017

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The human immunodeficiency virus type 1 ribosomal frameshifting site is an invariant sequence determinant and an important target for antiviral therapy

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Identification of residues of CXCR4 critical for human immunodeficiency virus coreceptor and chemokine receptor activities

7 January 2021

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7 January 2021

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Structure and function of the stimulatory RNAs involved in programmed eukaryotic-1 ribosomal frameshifting

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Mutational analysis of the "slippery-sequence" component of a coronavirus ribosomal frameshifting signal

7 January 2021

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An RNA pseudoknot and an optimal heptameric shift site are required for highly efficient ribosomal frameshifting on a retroviral messenger RNA

7 January 2021

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Characterization of RNA elements that regulate gag-pol ribosomal frameshifting in equine infectious anemia virus

7 January 2021

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Characterization of the frameshift stimulatory signal controlling a programmed -1 ribosomal frameshift in the human immunodeficiency virus type 1.

7 January 2021

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Structure of the RNA signal essential for translational frameshifting in HIV-1

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7 January 2021

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Proline residues within spacer peptide p1 are important for human immunodeficiency virus type 1 infectivity, protein processing, and genomic RNA dimer stability

7 January 2021

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7 January 2021

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Transfecting mammalian cells: optimization of critical parameters affecting calcium-phosphate precipitate formation

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In vitro evidence for a long range pseudoknot in the 5'-untranslated and matrix coding regions of HIV-1 genomic RNA

7 January 2021

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7 January 2021

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Contribution of the Gag-Pol transframe domain p6* and its coding sequence to morphogenesis and replication of human immunodeficiency virus type 1.

7 January 2021

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Changes in growth properties on passage in tissue culture of viruses derived from infectious molecular clones of HIV-1LAI, HIV-1MAL, and HIV-1ELI.

7 January 2021

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Replacement of the P1 amino acid of human immunodeficiency virus type 1 Gag processing sites can inhibit or enhance the rate of cleavage by the viral protease

7 January 2021

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Maintenance of the Gag/Gag-Pol ratio is important for human immunodeficiency virus type 1 RNA dimerization and viral infectivity

7 January 2021

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The conformation of the mature dimeric human immunodeficiency virus type 1 RNA genome requires packaging of pol protein.

7 January 2021

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Solution structure and thermodynamic investigation of the HIV-1 frameshift inducing element

7 January 2021

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Lentivirus replication and regulation

7 January 2021

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Analysis of natural variants of the human immunodeficiency virus type 1 gag-pol frameshift stem-loop structure

7 January 2021

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Fusion from without directed by human immunodeficiency virus particles

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.08.048

inferred from DOI database lookup

7 January 2021

based on heuristic

Role of Pr160gag-pol in mediating the selective incorporation of tRNA(Lys) into human immunodeficiency virus type 1 particles

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2005.08.048

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/J.VIROL.2005.08.048

1 reference

14 December 2017

1 reference