(Q45194007)

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Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment

scientific article published in December 2004

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment (English)

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

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Mark A Prendergast

5

object named as

Mark A Prendergast

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

author name string

Rachel L Self

1

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

Patrick J Mulholland

2

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

Barton R Harris

3

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

Avindra Nath

4

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

publication date

1 December 2004

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Europe PubMed Central

PubMed publication ID

10 December 2017

Alcoholism: Clinical and Experimental Research

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

28

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

12

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

1916-1924

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

Temporal relationships between HIV-1 Tat-induced neuronal degeneration, OX-42 immunoreactivity, reactive astrocytosis, and protein oxidation in the rat striatum.

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HIV-1 Tat causes apoptotic death and calcium homeostasis alterations in rat neurons.

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Methamphetamine potentiates HIV-1 Tat protein-mediated activation of redox-sensitive pathways in discrete regions of the brain

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HIV-1 Tat through phosphorylation of NMDA receptors potentiates glutamate excitotoxicity

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Evidence for a gender-related effect of alcoholism on brain volumes

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Post-insult activity is a major cause of delayed neuronal death in organotypic hippocampal slices exposed to glutamate

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Ethanol withdrawal hyper‐responsiveness mediated by NMDA receptors in spinal cord motor neurons

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Blood ethanol concentration profiles: a comparison between rats and mice

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Neuronal injury in hippocampus with human immunodeficiency virus transactivating protein, Tat.

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Septotemporal distribution of [3H]MK-801, [3H]AMPA and [3H]Kainate binding sites in the rat hippocampus

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Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression

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Transection of intrinsic polysynaptic pathways reduces N-methyl-D-aspartate neurotoxicity in hippocampal slice cultures

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NR2B subunit selective NMDA antagonists inhibit neurotoxic effect of alcohol-withdrawal in primary cultures of rat cortical neurones

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Increased sensitivity to NMDA is involved in alcohol-withdrawal induced cytotoxicity observed in primary cultures of cortical neurones chronically pre-treated with ethanol.

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Synergistic neurotoxicity by human immunodeficiency virus proteins Tat and gp120: protection by memantine

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HIV-1 Tat induces neuronal death via tumor necrosis factor-alpha and activation of non-N-methyl-D-aspartate receptors by a NFkappaB-independent mechanism

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Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-D-aspartate receptors

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Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-D-aspartate receptor

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The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site

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Identifiers

10.1097/01.ALC.0000148108.93782.05

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

10 December 2017

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