(Q44384607)

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Fluid flow shear stress stimulates human osteoblast proliferation and differentiation through multiple interacting and competing signal transduction pathways.

scientific article published in March 2003

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Europe PubMed Central

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2 November 2019

Fluid flow shear stress stimulates human osteoblast proliferation and differentiation through multiple interacting and competing signal transduction pathways (English)

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2 November 2019

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2 November 2019

author name string

Sonia Kapur

1

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2 November 2019

David J Baylink

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2 November 2019

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1 March 2003

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2 November 2019

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2 November 2019

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2 November 2019

3

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2 November 2019

241-251

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2 November 2019

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Pulsating fluid flow increases nitric oxide (NO) synthesis by osteocytes but not periosteal fibroblasts--correlation with prostaglandin upregulation

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Mechanical strain and fluid movement both activate extracellular regulated kinase (ERK) in osteoblast-like cells but via different signaling pathways

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The production of nitric oxide and prostaglandin E(2) by primary bone cells is shear stress dependent

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Involvement of different ion channels in osteoblasts' and osteocytes' early responses to mechanical strain

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Parathyroid hormone enhances fluid shear-induced [Ca2+]i signaling in osteoblastic cells through activation of mechanosensitive and voltage-sensitive Ca2+ channels

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Mitogen-activated protein kinases p42mapk and p44mapk are required for fibroblast proliferation

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Hypergravity stimulates collagen synthesis in human osteoblast-like cells: evidence for the involvement of p44/42 MAP-kinases (ERK 1/2)

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Fluid flow induction of cyclo-oxygenase 2 gene expression in osteoblasts is dependent on an extracellular signal-regulated kinase signaling pathway

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Osteopontin gene regulation by oscillatory fluid flow via intracellular calcium mobilization and activation of mitogen-activated protein kinase in MC3T3-E1 osteoblasts

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Proliferation and differentiation of human osteoblastic cells associated with differential activation of MAP kinases in response to epidermal growth factor, hypoxia, and mechanical stress in vitro

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Mechanotransduction: all signals point to cytoskeleton, matrix, and integrins

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Identifiers

10.1016/S8756-3282(02)00979-1

1 reference

Europe PubMed Central

PubMed publication ID

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2 November 2019

PubMed publication ID

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2 November 2019

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