(Q40977867)

English

Prevention of oxidant-induced cell death in Caco-2 colon carcinoma cells after inhibition of poly(ADP-ribose) polymerase and Ca2+ chelation: involvement of a common mechanism

scientific article published on January 1999

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scholarly article

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Europe PubMed Central

PubMed publication ID

25 September 2017

Prevention of oxidant-induced cell death in Caco-2 colon carcinoma cells after inhibition of poly(ADP-ribose) polymerase and Ca2+ chelation: involvement of a common mechanism (English)

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

1 reference

based on heuristic

inferred from title

colon carcinoma

1 reference

based on heuristic

inferred from title

author name string

Karczewski JM

1

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

Peters JG

2

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Europe PubMed Central

PubMed publication ID

25 September 2017

Noordhoek J

3

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Europe PubMed Central

PubMed publication ID

25 September 2017

language of work or name

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publication date

1 January 1999

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Europe PubMed Central

PubMed publication ID

25 September 2017

Biochemical Pharmacology

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Europe PubMed Central

PubMed publication ID

25 September 2017

57

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Europe PubMed Central

PubMed publication ID

25 September 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

19-26

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

Oxidative stress. Biochemistry and human disease

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Glutathione metabolism in Crohn's disease

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Oxygen radicals in ulcerative colitis

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Detection of oxygen radicals during reperfusion of intestinal cells in vitro

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Alterations in intracellular thiol homeostasis during the metabolism of menadione by isolated rat hepatocytes

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Menadione-induced cytotoxicity is associated with protein thiol oxidation and alteration in intracellular Ca2+ homeostasis

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Alterations of surface morphology caused by the metabolism of menadione in mammalian cells are associated with the oxidation of critical sulfhydryl groups in cytoskeletal proteins

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Menadione-induced bleb formation in hepatocytes is associated with the oxidation of thiol groups in actin

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Inhibition of hepatocyte plasma membrane Ca2+-ATPase activity by menadione metabolism and its restoration by thiols

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DNA breakage caused by hydrogen peroxide produced during the metabolism of 2-methyl-1,4-naphthoquinone (menadione) does not contribute to the cytotoxic action of the quinone

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Menadione-induced DNA fragmentation without 8-oxo-2′-deoxyguanosine formation in isolated rat hepatocytes

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Induction of dna damage by menadione (2-methyl-1,4-naphthoquinone) in primary cultures of rat hepatocytes

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DNA damage and lethal effects of hydrogen peroxide and menadione in Chinese hamster cells: distinct mechanisms are involved

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Quinone-induced DNA single strand breaks in rat hepatocytes and human chronic myelogenous leukaemic K562 cells

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Molecular and biochemical features of poly (ADP-ribose) metabolism

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Glutathione cycle activity and pyridine nucleotide levels in oxidant-induced injury of cells

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Oxidant injury of cells. DNA strand-breaks activate polyadenosine diphosphate-ribose polymerase and lead to depletion of nicotinamide adenine dinucleotide

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Hydrogen peroxide-induced injury of cells and its prevention by inhibitors of poly(ADP-ribose) polymerase

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Poly(ADP-ribose) polymerase inhibitors preserve oxidized nicotinamide adenine dinucleotide and adenosine 5'-triphosphate pools in DNA-damaged cells: mechanism of stimulation of unscheduled DNA synthesis

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Modulation of inflammatory arthritis by inhibition of poly(ADP ribose) polymerase

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Redox cycling and sulphydryl arylation; their relative importance in the mechanism of quinone cytotoxicity to isolated hepatocytes

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Toxicity determined in vitro by morphological alterations and neutral red absorption

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Methodologies for the application of monobromobimane to the simultaneous analysis of soluble and protein thiol components of biological systems

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Intracellular Ca2+ chelators prevent DNA damage and protect hepatoma 1C1C7 cells from quinone-induced cell killing

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Poly(ADP-ribose) polymerase: a molecular nick-sensor

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The role of proteases during apoptosis.

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Possible involvement of poly(ADP-ribosyl) polymerase in triggering stress-induced apoptosis.

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Formation and reduction of glutathione-protein mixed disulfides during oxidative stress. A study with isolated hepatocytes and menadione (2-methyl-1,4-naphthoquinone)

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Interaction with cellular ATP generating pathways mediates menadione-induced cytotoxicity in isolated rat hepatocytes

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Interconversion of NAD(H) to NADP(H). A cellular response to quinone-induced oxidative stress in isolated hepatocytes

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Inhibition of poly(ADP-ribose) polymerization preserves the glutathione pool and reverses cytotoxicity in hydrogen peroxide-treated lymphocytes

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Caco-2 cell metabolism of oxygen-derived radicals

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Correlation between cytosolic Ca2+ concentration and cytotoxicity in hepatocytes exposed to oxidative stress

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Stimulation of endogenous endonuclease activity in hepatocytes exposed to oxidative stress

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Identifiers

10.1016/S0006-2952(98)00286-X

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

25 September 2017

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