(Q40794732)

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The apoptotic regulatory protein ARC (apoptosis repressor with caspase recruitment domain) prevents oxidant stress-mediated cell death by preserving mitochondrial function

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Europe PubMed Central

PubMed publication ID

23 September 2017

The apoptotic regulatory protein ARC (apoptosis repressor with caspase recruitment domain) prevents oxidant stress-mediated cell death by preserving mitochondrial function (English)

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

apoptotic process

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author name string

Neuss M

1

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Monticone R

2

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Lundberg MS

3

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Chesley AT

4

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Fleck E

5

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

Crow MT

6

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Europe PubMed Central

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23 September 2017

language of work or name

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publication date

3 July 2001

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Europe PubMed Central

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23 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

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23 September 2017

276

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Europe PubMed Central

PubMed publication ID

23 September 2017

36

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Europe PubMed Central

PubMed publication ID

23 September 2017

33915-33922

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Europe PubMed Central

PubMed publication ID

23 September 2017

The modular nature of apoptotic signaling proteins

1 reference

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Apoptosis by death factor

1 reference

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The CARD domain: a new apoptotic signalling motif

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An induced proximity model for caspase-8 activation.

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RAIDD is a new 'death' adaptor molecule

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

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21 January 2018

IAP family proteins--suppressors of apoptosis

1 reference

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21 January 2018

RICK, a novel protein kinase containing a caspase recruitment domain, interacts with CLARP and regulates CD95-mediated apoptosis

1 reference

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Identification of CARDIAK, a RIP-like kinase that associates with caspase-1

1 reference

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CIPER, a novel NF kappaB-activating protein containing a caspase recruitment domain with homology to Herpesvirus-2 protein E10

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mE10, a novel caspase recruitment domain-containing proapoptotic molecule

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21 January 2018

CLAP, a novel caspase recruitment domain-containing protein in the tumor necrosis factor receptor pathway, regulates NF-kappaB activation and apoptosis

1 reference

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21 January 2018

ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases

1 reference

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21 January 2018

ARC inhibits cytochrome c release from mitochondria and protects against hypoxia-induced apoptosis in heart-derived H9c2 cells

1 reference

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Direct measurement of free radical generation following reperfusion of ischemic myocardium

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Direct evidence that oxygen-derived free radicals contribute to postischemic myocardial dysfunction in the intact dog.

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Alternative splicing determines the intracellular localization of the novel nuclear protein Nop30 and its interaction with the splicing factor SRp30c

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A simplified system for generating recombinant adenoviruses

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Nod1, an Apaf-1-like activator of caspase-9 and nuclear factor-kappaB

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Human CARD4 protein is a novel CED-4/Apaf-1 cell death family member that activates NF-kappaB

1 reference

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The c-IAP-1 and c-IAP-2 proteins are direct inhibitors of specific caspases

1 reference

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RIP2 is a novel NF-kappaB-activating and cell death-inducing kinase

1 reference

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21 January 2018

The mitochondrial permeability transition.

1 reference

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21 January 2018

Mitochondrial control of cell death

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Modulation of the mitochondrial permeability transition pore by pyridine nucleotides and dithiol oxidation at two separate sites.

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Opening of the mitochondrial permeability transition pore by uncoupling or inorganic phosphate in the presence of Ca2+ is dependent on mitochondrial-generated reactive oxygen species.

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Role of Ca2+ in toxic cell killing

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Differential effects of bcl-2 on cell death triggered under ATP-depleting conditions.

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Protection against hydrogen peroxide cytotoxicity in rat-1 fibroblasts provided by the oncoprotein Bcl-2: maintenance of calcium homoeostasis is secondary to the effect of Bcl-2 on cellular glutathione

1 reference

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21 January 2018

Mitochondria and cell death. Mechanistic aspects and methodological issues

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The mitochondrial permeability transition in cell death: a common mechanism in necrosis, apoptosis and autophagy

1 reference

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21 January 2018

Identifiers

10.1074/JBC.M104080200

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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