(Q40504123)

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The glucocorticoid-induced gene tdag8 encodes a pro-apoptotic G protein-coupled receptor whose activation promotes glucocorticoid-induced apoptosis

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Europe PubMed Central

PubMed publication ID

21 September 2017

The glucocorticoid-induced gene tdag8 encodes a pro-apoptotic G protein-coupled receptor whose activation promotes glucocorticoid-induced apoptosis (English)

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

G protein-coupled receptor

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apoptotic process

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author name string

Michael H Malone

1

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

Zhengqi Wang

2

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

Clark W Distelhorst

3

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Europe PubMed Central

PubMed publication ID

21 September 2017

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publication date

12 October 2004

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Europe PubMed Central

PubMed publication ID

21 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

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21 September 2017

279

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Europe PubMed Central

PubMed publication ID

21 September 2017

51

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

52850-52859

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

Adrenocorticotropic hormone- and cortisone-induced regression of lymphoid tumors in man; a preliminary report

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Immune homeostasis requires several biologic factors including glucocorticoid hormones

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Crosstalk pathway for inhibition of glucocorticoid-induced apoptosis by T cell receptor signaling

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Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation

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Glucocorticoids in T cell development and function*.

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Glucocorticoids activate a suicide process in thymocytes through an elevation of cytosolic Ca2+ concentration

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Chromatin cleavage in apoptosis: association with condensed chromatin morphology and dependence on macromolecular synthesis.

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Mechanisms of steroid resistance.

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Glucocorticoid receptors in lymphoma cells in culture: relationship to glucocorticoid killing activity.

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Receptors from glucocorticoid-sensitive lymphoma cells and two clases of insensitive clones: physical and DNA-binding properties

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Recent insights into the mechanism of glucocorticosteroid-induced apoptosis

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Glucocorticoid-induced apoptosis and glucocorticoid resistance: molecular mechanisms and clinical relevance.

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Glucocorticoid-induced apoptosis of thymocytes: requirement of proteasome-dependent mitochondrial activity

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Elevated glutathione S-transferase gene expression is an early event during steroid-induced lymphocyte apoptosis.

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Deficiency in Bak and Bax perturbs thymic selection and lymphoid homeostasis.

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Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics

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Microarray analysis uncovers the induction of the proapoptotic BH3-only protein Bim in multiple models of glucocorticoid-induced apoptosis

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Identification of novel isoforms of the BH3 domain protein Bim which directly activate Bax to trigger apoptosis

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21 January 2018

The pro-apoptotic protein Bim is a convergence point for cAMP/protein kinase A- and glucocorticoid-promoted apoptosis of lymphoid cells

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Rescue of thymocytes and T cell hybridomas from glucocorticoid-induced apoptosis by stimulation via the T cell receptor/CD3 complex: a possiblein vitro model for positive selection of the T cell repertoire

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Dexamethasone-induced Gene 2 (dig2) Is a Novel Pro-survival Stress Gene Induced Rapidly by Diverse Apoptotic Signals

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Significance analysis of microarrays applied to the ionizing radiation response

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Identification of a molecular target of psychosine and its role in globoid cell formation

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Transient expression of wild-type or mitochondrially targeted Bcl-2 induces apoptosis, whereas transient expression of endoplasmic reticulum-targeted Bcl-2 is protective against Bax-induced cell death

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21 January 2018

Bim: a novel member of the Bcl-2 family that promotes apoptosis

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Sphingosylphosphorylcholine and lysophosphatidylcholine are ligands for the G protein-coupled receptor GPR4

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RETRACTED: Lysophosphatidylcholine as a ligand for the immunoregulatory receptor G2A.

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Identifiers

10.1074/JBC.M408040200

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

21 September 2017

ResearchGate publication ID

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