(Q40150418)

English

Poly-(ADP-ribose) polymerase-1 (Parp-1) binds in a sequence-specific manner at the Bcl-6 locus and contributes to the regulation of Bcl-6 transcription

scientific article published on 2 April 2007

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scholarly article

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Europe PubMed Central

PubMed publication ID

17 September 2017

Poly-(ADP-ribose) polymerase-1 (Parp-1) binds in a sequence-specific manner at the Bcl-6 locus and contributes to the regulation of Bcl-6 transcription (English)

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

4

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

author name string

H E Ambrose

1

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

V Papadopoulou

2

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

R W Beswick

3

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

language of work or name

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publication date

2 April 2007

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

26

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

42

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

6244-6252

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

Activation of Reg gene, a gene for insulin-producing beta -cell regeneration: poly(ADP-ribose) polymerase binds Reg promoter and regulates the transcription by autopoly(ADP-ribosyl)ation

1 reference

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The PARP superfamily

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Differential regulation of CXC ligand 1 transcription in melanoma cell lines by poly(ADP-ribose) polymerase-1.

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Acetylation inactivates the transcriptional repressor BCL6.

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Small deletions occur in highly conserved regions of the LAZ3/BCL6 major translocation cluster in one case of non-Hodgkin's lymphoma without 3q27 translocation

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A B-cell-specific DNA recombination complex

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DNA Double-Strand Breaks in Immunoglobulin Genes Undergoing Somatic Hypermutation

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Poly(ADP-ribose) polymerase binds with transcription enhancer factor 1 to MCAT1 elements to regulate muscle-specific transcription

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Poly(ADP-ribose) polymerase is a B-MYB coactivator

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BCL-6, a POZ/zinc-finger protein, is a sequence-specific transcriptional repressor

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Analysis of multiple genomic sequence alignments: a web resource, online tools, and lessons learned from analysis of mammalian SCL loci.

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A peptide aptamer to antagonize BCL-6 function.

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ETO protein of t(8;21) AML is a corepressor for Bcl-6 B-cell lymphoma oncoprotein.

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Control of inflammation, cytokine expression, and germinal center formation by BCL-6

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MTA3 and the Mi-2/NuRD complex regulate cell fate during B lymphocyte differentiation

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Long-range comparison of human and mouse SCL loci: localized regions of sensitivity to restriction endonucleases correspond precisely with peaks of conserved noncoding sequences

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The enzymatic and DNA binding activity of PARP-1 are not required for NF-kappa B coactivator function

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Analysis of nucleotide sequence-dependent DNA binding of poly(ADP-ribose) polymerase in a purified system

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PARP-1, PARP-2 and ATM in the DNA damage response: functional synergy in mouse development

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The BCL-6 POZ domain and other POZ domains interact with the co-repressors N-CoR and SMRT.

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A topoisomerase IIbeta-mediated dsDNA break required for regulated transcription.

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Activating the PARP-1 sensor component of the groucho/ TLE1 corepressor complex mediates a CaMKinase IIdelta-dependent neurogenic gene activation pathway

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Identification of negative regulatory regions within the first exon and intron of the BCL6 gene.

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NAD+-dependent modulation of chromatin structure and transcription by nucleosome binding properties of PARP-1.

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PARP goes transcription

1 reference

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Regulation of poly(ADP-ribose) polymerase-1 by DNA structure-specific binding

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Two new isotype-specific switching activities detected for Ig class switching

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BCL6 is regulated by p53 through a response element frequently disrupted in B-cell non-Hodgkin lymphoma.

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XRCC1 is specifically associated with poly(ADP-ribose) polymerase and negatively regulates its activity following DNA damage

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A role for poly(ADP-ribose) polymerase in the transcriptional regulation of the melanoma growth stimulatory activity (CXCL1) gene expression

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The histone variant mH2A1.1 interferes with transcription by down-regulating PARP-1 enzymatic activity

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Mutations of the BCL6 proto-oncogene disrupt its negative autoregulation in diffuse large B-cell lymphoma.

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PARP-1 determines specificity in a retinoid signaling pathway via direct modulation of mediator

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Transcription. Puffing with PARP.

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Poly(ADP-ribosyl)ation of polynucleosomes causes relaxation of chromatin structure

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Specific peptide interference reveals BCL6 transcriptional and oncogenic mechanisms in B-cell lymphoma cells.

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Poly(ADP-ribosyl)ated chromatin domains: access granted

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TdT-accessible breaks are scattered over the immunoglobulin V domain in a constitutively hypermutating B cell line

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Role of poly(ADP-ribose) formation in DNA repair

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Rapid detection of octamer binding proteins with 'mini-extracts', prepared from a small number of cells

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Poly(ADP-ribose): novel functions for an old molecule

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PARP-1 binds E2F-1 independently of its DNA binding and catalytic domains, and acts as a novel coactivator of E2F-1-mediated transcription during re-entry of quiescent cells into S phase

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STAT5 represses BCL6 expression by binding to a regulatory region frequently mutated in lymphomas

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Negative autoregulation of BCL-6 is bypassed by genetic alterations in diffuse large B cell lymphomas

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The BCL-6 proto-oncogene controls germinal-centre formation and Th2-type inflammation

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Alterations of a zinc finger-encoding gene, BCL-6, in diffuse large-cell lymphoma

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Sequence-specific binding of poly(ADP-ribose) polymerase-1 to the human T cell leukemia virus type-I tax responsive element

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(ADP-ribose)n participates in DNA excision repair

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Poly(ADP-ribosyl)ation reactions in the regulation of nuclear functions

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Transcription. Gene expression needs a break to unwind before carrying on

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Rearrangement of the bcl-6 gene as a prognostic marker in diffuse large-cell lymphoma

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Specific binding of poly(ADP-ribose) polymerase-1 to cruciform hairpins

1 reference

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Transcriptional repression by binding of poly(ADP-ribose) polymerase to promoter sequences

1 reference

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Chromosomal translocations cause deregulated BCL6 expression by promoter substitution in B cell lymphoma.

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inferred from DOI database lookup

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10.1038/SJ.ONC.1210434

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

17 September 2017

ResearchGate publication ID

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