(Q33844558)

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Long term lithium treatment suppresses p53 and Bax expression but increases Bcl-2 expression. A prominent role in neuroprotection against excitotoxicity

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Europe PubMed Central

PubMed publication ID

29 July 2017

Long term lithium treatment suppresses p53 and Bax expression but increases Bcl-2 expression. A prominent role in neuroprotection against excitotoxicity (English)

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

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Chen RW

1

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Europe PubMed Central

PubMed publication ID

29 July 2017

Chuang DM

2

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Europe PubMed Central

PubMed publication ID

29 July 2017

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publication date

1 March 1999

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Europe PubMed Central

PubMed publication ID

29 July 2017

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

29 July 2017

274

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Europe PubMed Central

PubMed publication ID

29 July 2017

10

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Europe PubMed Central

PubMed publication ID

29 July 2017

6039-6042

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Europe PubMed Central

PubMed publication ID

29 July 2017

G proteins: implications for psychiatry

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21 January 2018

CNS signal transduction in the pathophysiology and pharmacotherapy of affective disorders and schizophrenia

1 reference

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21 January 2018

AP-1 and NF-kappaB stimulated by carbachol in human neuroblastoma SH-SY5Y cells are differentially sensitive to inhibition by lithium

1 reference

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21 January 2018

Lithium stimulates gene expression through the AP-1 transcription factor pathway

1 reference

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21 January 2018

Chronic lithium treatment robustly protects neurons in the central nervous system against excitotoxicity by inhibiting N-methyl-D-aspartate receptor-mediated calcium influx

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21 January 2018

Neuroprotective effects of chronic lithium on focal cerebral ischemia in rats

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21 January 2018

Bax directly induces release of cytochrome c from isolated mitochondria

1 reference

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21 January 2018

Apoptosis induction by caspase-8 is amplified through the mitochondrial release of cytochrome c.

1 reference

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21 January 2018

Mechanisms controlling cellular suicide: role of Bcl-2 and caspases

1 reference

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21 January 2018

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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21 January 2018

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

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21 January 2018

Inhibition of Bax channel-forming activity by Bcl-2

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21 January 2018

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

1 reference

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21 January 2018

Activation of a caspase 3-related cysteine protease is required for glutamate-mediated apoptosis of cultured cerebellar granule neurons

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21 January 2018

Bcl-2 inhibits the mitochondrial release of an apoptogenic protease

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21 January 2018

Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation

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21 January 2018

The Bcl-2 protein family: arbiters of cell survival

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21 January 2018

Bcl-2 down-regulates the activity of transcription factor NF-kappaB induced upon apoptosis

1 reference

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21 January 2018

Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2+ fluxes

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21 January 2018

A molecular mechanism for the effect of lithium on development

1 reference

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21 January 2018

A common denominator linking glycogen metabolism, nuclear oncogenes and development

1 reference

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21 January 2018

Identifiers

10.1074/JBC.274.10.6039

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

29 July 2017

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