(Q28586997)

31 January 2017

title

Role of ERO1-alpha-mediated stimulation of inositol 1,4,5-triphosphate receptor activity in endoplasmic reticulum stress-induced apoptosis (English)

1 reference

DNA-damage inducible transcript 3

31 January 2017

main subject

endoplasmic reticulum

0 references

apoptotic process

0 references

1 reference

Inositol 1,4,5-trisphosphate receptor 1

GOA release 2020-03-11

1 reference

Endoplasmic reticulum oxidoreductase 1 alpha

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

Heather Harding

4

0 references

David Ron

object named as

David Ron

5

0 references

King-Tung Chin

3

object named as

King-Tung Chin

1 reference

31 January 2017

Gang Li

1

1 reference

31 January 2017

Marco Mongillo

2

1 reference

31 January 2017

Andrew R. Marks

6

1 reference

31 January 2017

Ira Tabas

7

1 reference

31 January 2017

language of work or name

English

0 references

publication date

21 September 2009

1 reference

31 January 2017

published in

Journal of Cell Biology

1 reference

31 January 2017

volume

186

1 reference

31 January 2017

issue

6

1 reference

31 January 2017

page(s)

783–792

1 reference

describes a project that uses

31 January 2017

ImageJ

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC2753154/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

cites work

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Endoplasmic reticulum stress induces calcium-dependent permeability transition, mitochondrial outer membrane permeabilization and apoptosis

17 March 2017

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Subtype-specific and ER lumenal environment-dependent regulation of inositol 1,4,5-trisphosphate receptor type 1 by ERp44

17 March 2017

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CHOP induces death by promoting protein synthesis and oxidation in the stressed endoplasmic reticulum

17 March 2017

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Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes

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ER stress triggers apoptosis by activating BH3-only protein Bim

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Mechanisms and consequences of macrophage apoptosis in atherosclerosis

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Chop deletion reduces oxidative stress, improves beta cell function, and promotes cell survival in multiple mouse models of diabetes

29 September 2017

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Forkhead transcription factors (FoxOs) promote apoptosis of insulin-resistant macrophages during cholesterol-induced endoplasmic reticulum stress

29 September 2017

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Signal transducer and activator of transcription-1 is critical for apoptosis in macrophages subjected to endoplasmic reticulum stress in vitro and in advanced atherosclerotic lesions in vivo.

29 September 2017

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29 September 2017

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Cholesterol-induced macrophage apoptosis requires ER stress pathways and engagement of the type A scavenger receptor

29 September 2017

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Enrichment of endoplasmic reticulum with cholesterol inhibits sarcoplasmic-endoplasmic reticulum calcium ATPase-2b activity in parallel with increased order of membrane lipids: implications for depletion of endoplasmic reticulum calcium stores and a

29 September 2017

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Ca2+-dependent redox modulation of SERCA 2b by ERp57.

29 September 2017

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Cytochrome c binds to inositol (1,4,5) trisphosphate receptors, amplifying calcium-dependent apoptosis

29 September 2017

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T cells deficient in inositol 1,4,5-trisphosphate receptor are resistant to apoptosis

29 September 2017

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Lymphocyte apoptosis: mediation by increased type 3 inositol 1,4,5-trisphosphate receptor

29 September 2017

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Complex regulation of heat shock- and glucose-responsive genes in human cells

29 September 2017

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Free cholesterol loading of macrophages induces apoptosis involving the fas pathway.

29 September 2017

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Effects of redox potential and Ca2+ on the inositol 1,4,5-trisphosphate receptor L3-1 loop region: implications for receptor regulation.

3 June 2018

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CHOP is involved in neuronal apoptosis induced by neurotrophic factor deprivation

27 November 2018

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Macrophage insulin receptor deficiency increases ER stress-induced apoptosis and necrotic core formation in advanced atherosclerotic lesions.

27 November 2018

1 reference

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The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages

27 November 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2753154

Free cholesterol loading of macrophages is associated with widespread mitochondrial dysfunction and activation of the mitochondrial apoptosis pathway

27 November 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2753154

Inositol-1,4,5-trisphosphate receptor-mediated Ca mobilization is not required for cerebellar long-term depression in reduced preparations

27 November 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2753154

The Phenotype of Inflammatory Macrophages Is Stimulus Dependent: Implications for the Nature of the Inflammatory Response

27 November 2018

1 reference

https://pubmed.ncbi.nlm.nih.gov/19752026

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Characterization of responses of isolated rat hepatocytes to ATP and ADP

1 reference

https://pubmed.ncbi.nlm.nih.gov/19752026

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1083/JCB.200904060

1 reference

1 reference

1 reference

1 reference