(Q28278844)

13

Enza Maria Valente

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Frank Reimann

Frank Reimann

2

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Fiona Gribble

Fiona M Gribble

18

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C Geoffrey Woods

19

C Geoffrey Woods

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author name string

James J Cox

1

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Adeline K Nicholas

3

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Gemma Thornton

4

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Emma Roberts

5

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Kelly Springell

6

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Gulshan Karbani

7

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Hussain Jafri

8

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Jovaria Mannan

9

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Yasmin Raashid

10

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Henan Hamamy

12

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Shaun Gorman

14

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Richard Williams

15

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Duncan P McHale

16

0 references

John N Wood

Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons

17

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language of work or name

English

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publication date

14 December 2006

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published in

Nature

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volume

444

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issue

7121

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page(s)

894-8

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cites work

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Indifference rather than insensitivity to pain

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Roles of tetrodotoxin (TTX)-sensitive Na+ current, TTX-resistant Na+ current, and Ca2+ current in the action potentials of nociceptive sensory neurons.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

A novel tetrodotoxin-sensitive, voltage-gated sodium channel expressed in rat and human dorsal root ganglia.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Structure and functional expression of a new member of the tetrodotoxin-sensitive voltage-activated sodium channel family from human neuroendocrine cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Sporadic onset of erythermalgia: a gain-of-function mutation in Nav1.7.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Early nonsense: mRNA decay solves a translational problem

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

https://pubmed.ncbi.nlm.nih.gov/17167479

1 reference

12 December 2020

Expression of alternatively spliced sodium channel alpha-subunit genes. Unique splicing patterns are observed in dorsal root ganglia

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

The role of sodium channels in chronic inflammatory and neuropathic pain

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Sodium channel beta subunits: anything but auxiliary

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Spinal sensory neurons express multiple sodium channel alpha-subunit mRNAs

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Dominantly transmitted congenital indifference to pain

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Molecular genetic approaches to nociceptor development and function.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Not 'indifference to pain' but varieties of hereditary sensory and autonomic neuropathy

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020

Congenital insensitivity to pain: an update

1 reference

https://pubmed.ncbi.nlm.nih.gov/17167479

12 December 2020