(Q28267232)

6

object named as

L M Neckers

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author name string

W G An

1

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M Kanekal

2

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M C Simon

3

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E Maltepe

Hypoxia induces accumulation of p53 protein, but activation of a G1-phase checkpoint by low-oxygen conditions is independent of p53 status.

4

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language of work or name

English

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publication date

26 March 1998

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published in

Nature

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volume

392

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issue

6674

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page(s)

405-8

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cites work

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7 January 2021

Regulation of the erythropoietin gene: evidence that the oxygen sensor is a heme protein

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Absolute requirement of aryl hydrocarbon receptor nuclear translocator protein for gene activation by hypoxia

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Functional interference between hypoxia and dioxin signal transduction pathways: competition for recruitment of the Arnt transcription factor

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The hypoxic response: huffing and HIFing

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Transcriptional regulation of the rat vascular endothelial growth factor gene by hypoxia

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Cloning of a Factor Required for Activity of the Ah (Dioxin) Receptor

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Abnormal angiogenesis and responses to glucose and oxygen deprivation in mice lacking the protein ARNT.

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General involvement of hypoxia-inducible factor 1 in transcriptional response to hypoxia

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Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

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7 January 2021

Purification and characterization of hypoxia-inducible factor 1

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The role of the aryl hydrocarbon receptor nuclear translocator (ARNT) in hypoxic induction of gene expression. Studies in ARNT-deficient cells

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Activation of hypoxia-inducible factor 1alpha: posttranscriptional regulation and conformational change by recruitment of the Arnt transcription factor

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Geldanamycin-stimulated destabilization of mutated p53 is mediated by the proteasome in vivo

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A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells

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An essential role for p300/CBP in the cellular response to hypoxia

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Activation of hypoxia-inducible transcription factor depends primarily upon redox-sensitive stabilization of its alpha subunit

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WAF1, a potential mediator of p53 tumor suppression

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