Wikidata

(Q31149957)

English

The coding sequence of the hemolytically inactive C4A6 allotype of human complement component C4 reveals that a single arginine to tryptophan substitution at beta-chain residue 458 is the likely cause of the defect

scientific article published on May 1, 1992

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title
The coding sequence of the hemolytically inactive C4A6 allotype of human complement component C4 reveals that a single arginine to tryptophan substitution at beta-chain residue 458 is the likely cause of the defect (English)
1 reference
stated in
Europe PubMed Central
PubMed publication ID
1573268
retrieved
29 June 2017

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