Eosinophilia: Difference between revisions

Hypereosinophilia may occur in the setting of damage to a single specific organ due to a massive infiltration by eosinophils. This disorder is sub-classified based on the organ involved and is not considered to be a form of primary hypereosinophilia, secondary hypereosinophilia, or the idiopathic hypereosinophilic syndrome because: '''a)''' the eosinophils associated with the disorder have not been shown to be clonal in nature; '''b)''' a reason for the increase in blood eosinophils has not been determined; '''c)''' organ damage has not been shown to be due to eosinophils; and '''d)''' the disorder in each individual case typically is limited to the afflicted organ. Examples of organ-restricted hypereosinopiliahypereosinophilia include [[eosinophilic myocarditis]], [[eosinophilic esophagitis]], [[eosinophilic gastroenteritis]], [[eosinophilic cystitis]], [[eosinophilic pneumonia]], [[eosinophilic fasciitis]], [[eosinophilic folliculitis]], [[eosinophilic cellulitis]], [[eosinophilic vasculitis]], and [[eosinophilic ulcer of the oral mucosa]]. Other examples of organ-restricted hepereosinophilia include those involving the heart, kidney, liver, colon, [[pulmonary pleurae]], [[peritoneum]], fat tissue, [[myometrium]], and [[synovia]].<ref name="pmid22460074"/>